Q
Qingwei Huo
Researcher at Guangzhou University of Chinese Medicine
Publications - 3
Citations - 54
Qingwei Huo is an academic researcher from Guangzhou University of Chinese Medicine. The author has contributed to research in topics: Inhibitory postsynaptic potential & GABAergic. The author has an hindex of 2, co-authored 3 publications receiving 11 citations.
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Journal ArticleDOI
Enhancing GABAergic signaling ameliorates aberrant gamma oscillations of olfactory bulb in AD mouse models
Ming Chen,Ming Chen,Yunan Chen,Qingwei Huo,Lei Wang,Shuyi Tan,Afzal Misrani,Jinxiang Jiang,Jian Chen,Shiyuan Chen,Jia-wei Zhang,Sidra Tabassum,Jichen Wang,Xi Chen,Cheng Long,Li Yang +15 more
TL;DR: In this article, the authors conducted in vivo measurement of local field potential (LFP) with a combination of electroolfactogram (EOG), whole-cell patch and field recordings to evaluate oscillatory and synaptic function and pharmacological regulation in the olfactory pathway.
Journal ArticleDOI
Systemic LPS-induced microglial activation results in increased GABAergic tone: A mechanism of protection against neuroinflammation in the medial prefrontal cortex in mice.
Jinxiang Jiang,Binliang Tang,Lei Wang,Qingwei Huo,Shuyi Tan,Afzal Misrani,Yuanyuan Han,Hui-dong Li,Hai-Dong Hu,Jichen Wang,Tinghui Cheng,Sidra Tabassum,Ming Chen,Wenyuan Xie,Cheng Long,Li Yang +15 more
TL;DR: In this article, the authors report that acute neuroinflammation induced by intraperitoneal injection of lipopolysaccharide (LPS) results in cell-type-specific increases in inhibitory postsynaptic currents in the glutamatergic, but not the GABAergic, neurons of medial prefrontal cortex (mPFC), coinciding with excessive microglial activation.
Posted ContentDOI
Microglial activation results in neuron-type-specific increase in mPFC GABAergic transmission and abnormal behavior in mice
Binliang Tang,Binliang Tang,Jinxiang Jiang,Lei Wang,Afzal Misrani,Qingwei Huo,Yuanyuan Han,Cheng Long,Li Yang +8 more
TL;DR: A novel GABAergic signaling pathway that might be targeted therapeutically to treat neuroinflammation-induced abnormal synaptic activity and associated aberrant behavior is revealed.