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Qiongmei Gao

Researcher at Shanghai Jiao Tong University

Publications -  5
Citations -  58

Qiongmei Gao is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Type 2 diabetes & Diabetes mellitus. The author has an hindex of 2, co-authored 3 publications receiving 5 citations.

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Decreased Abundance of Akkermansia muciniphila Leads to the Impairment of Insulin Secretion and Glucose Homeostasis in Lean Type 2 Diabetes

TL;DR: In this paper, a metagenomic and targeted metabolomic analysis was conducted in 182 lean and abdominally obese individuals with and without newly diagnosed Type 2 diabetes (T2D) and showed that the abundance of Akkermansia muciniphila significantly decreases in lean individuals with type 2 diabetes than without T2D.
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Elevated Serum Level of Cytokeratin 18 M65ED Is an Independent Indicator of Cardiometabolic Disorders.

TL;DR: Elevated levels of CK18 M65ED, a sensitive cell death marker, were independently and positively correlated with cardiometabolic disorders, even after the adjustment for the presence of NAFLD and other cardiovascular risk factors.
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FGF21/adiponectin ratio predicts deterioration in glycemia: a 4.6-year prospective study in China.

TL;DR: Wang et al. as discussed by the authors investigated the relationship between the fibroblast growth factor (FGF) 21-adiponectin pathway and type 2 diabetes in a prospective cohort study.
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Effects of Exercise Intervention on Type 2 Diabetes Patients With Abdominal Obesity and Low Thigh Circumference (EXTEND): Study Protocol for a Randomized Controlled Trial

TL;DR: The results will provide a comprehensive evaluation of the physiological effects of exercise, and reveal the role of the microbiota-gut-brain axis in exercise-induced metabolic benefits to diabetes.
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Combined HASPIN and mTOR inhibition is synergistic against KRAS-driven carcinomas

TL;DR: In this article , a combination of mTOR and HASPIN inhibitors was used to prevent the phosphorylation of H3 histones, exacerbating mitotic catastrophe and DNA damage in tumor cell lines with KRAS mutations and this effect is due in part to a reduction in VRK1.