R
R E Kessler
Researcher at Rockefeller University
Publications - 5
Citations - 559
R E Kessler is an academic researcher from Rockefeller University. The author has contributed to research in topics: Lipoteichoic acid & Gel electrophoresis. The author has an hindex of 5, co-authored 5 publications receiving 533 citations.
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Journal ArticleDOI
Growth characteristics of group A streptococci in a new chemically defined medium.
I van de Rijn,R E Kessler +1 more
TL;DR: A new chemically defined medium for the growth of group A streptococci has been formulated and the production of virulence factors and extracellular enzymes during growth in this new medium was comparable to that in complex media.
Journal ArticleDOI
Characterization and localization of the enzymatic deacylation of lipoteichoic acid in group A streptococci.
TL;DR: The chemical data suggest that the enzyme was lipolytic in nature; that is, the conversion of LTA to dLTA was the result of cleavage of the ester linkages between the fatty acids and the remainder of the LTA molecule.
Journal ArticleDOI
Chemical Analysis of Changes in Membrane Composition During Growth of Streptococcus pyogenes
Ivo van de Rijn,R E Kessler +1 more
TL;DR: Results provide evidence that the composition of membranes of S. pyogenes does not remain constant throughout the growth phases of the culture.
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Quantitative immunoelectrophoretic analysis of Streptococcus pyogenes membrane.
R E Kessler,Ivo van de Rijn +1 more
TL;DR: The results from absorption experiments suggested that the exposure of outer protoplast surface antigens was greater on protoplasts from exponential-phase cells than on those from stationary- phase cells, even when found in increased amounts in the latter.
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Effects of penicillin on group A streptococci: loss of viability appears to precede stimulation of release of lipoteichoic acid.
R E Kessler,I van de Rijn +1 more
TL;DR: Although decreases in viability were observed within 15 min after addition of penicillin, culture mass and LTA content did not appear to be affected until after 30 min, which is interpreted to indicate that the stimulation of release of LTA and dLTA in response toPenicillin is secondary to the killing event.