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Ralph J. Delfino
Researcher at University of California, Irvine
Publications - 122
Citations - 10447
Ralph J. Delfino is an academic researcher from University of California, Irvine. The author has contributed to research in topics: Environmental exposure & Population. The author has an hindex of 54, co-authored 122 publications receiving 9659 citations. Previous affiliations of Ralph J. Delfino include San Diego State University & University of California.
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Journal ArticleDOI
Potential role of ultrafine particles in associations between airborne particle mass and cardiovascular health.
TL;DR: The need for and methods of UFP exposure assessment are discussed, which may lead to systemic inflammation through oxidative stress responses to reactive oxygen species and thereby promote the progression of atherosclerosis and precipitate acute cardiovascular responses ranging from increased blood pressure to myocardial infarction.
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Exposure assessment for atmospheric ultrafine particles (UFPs) and implications in epidemiologic research
TL;DR: The present review provides a detailed discussion of key characteristics of UFPs, their sources and formation mechanisms, and methodologic approaches to assessing population exposures.
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The relationship of respiratory and cardiovascular hospital admissions to the southern California wildfires of 2003.
Ralph J. Delfino,Sean S Brummel,Jun Wu,Hal S. Stern,Bart Ostro,Michael Lipsett,Arthur M. Winer,Donald H. Street,Lixia Zhang,Thomas Tjoa,Daniel L. Gillen +10 more
TL;DR: Wildfire-related PM2.5 led to increased respiratory hospital admissions, especially asthma, suggesting that better preventive measures are required to reduce morbidity among vulnerable populations.
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Circulating biomarkers of inflammation, antioxidant activity, and platelet activation are associated with primary combustion aerosols in subjects with coronary artery disease.
Ralph J. Delfino,Norbert Staimer,Thomas Tjoa,Andrea Polidori,Mohammad Arhami,Daniel L. Gillen,Micheal T. Kleinman,Nosratola D. Vaziri,John C. Longhurst,Frank Zaldivar,Constantinos Sioutas +10 more
TL;DR: It is suggested that traffic emission sources of OCpri and quasi-ultrafine particles lead to increased systemic inflammation and platelet activation and decreased antioxidant enzyme activity in elderly people with CAD.
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Epidemiologic evidence for asthma and exposure to air toxics: linkages between occupational, indoor, and community air pollution research.
TL;DR: Outdoor ambient air pollutant exposures in communities are relevant to the acute exacerbation and possibly the onset of asthma, but the complexity of pollutant mixtures and etiologic heterogeneity of asthma has made it difficult to identify causal components in those mixtures.