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Regina Hollweck

Researcher at Technische Universität München

Publications -  21
Citations -  1310

Regina Hollweck is an academic researcher from Technische Universität München. The author has contributed to research in topics: Ischemia & Lacidipine. The author has an hindex of 15, co-authored 21 publications receiving 1219 citations.

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Comparison between breast volume measurement using 3D surface imaging and classical techniques

TL;DR: Compared with three classical methods of breast volume calculation, 3D scanning provides acceptable accuracy for breast volume measurements, better spatial interpretation of the anatomical area to be operated on, non-invasiveness, and good patient tolerance.
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Effect of ozone on oral cells compared with established antimicrobials.

TL;DR: Aqueous ozone revealed the highest level of biocompatibility of the tested antiseptics, compared with the antibiotic, metronidazole, over 24 h.
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New aspects of breast volume measurement using 3-dimensional surface imaging.

TL;DR: Three-dimensional scanning of the body surface allows reproducible and objective assessment of the complex breast region but requires further investigation before clinical application, and represents a sufficiently precise and accurate method to guarantee objective and exact recording.
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Baseline values but not treatment-induced changes in carotid intima-media thickness predict incident cardiovascular events in treated hypertensive patients: findings in the European Lacidipine Study on Atherosclerosis (ELSA).

TL;DR: ELSA shows that carotid intima-media thickening and plaques are important added risks of cardiovascular outcomes in a treated hypertensive population independently of blood pressure and traditional risk factors, but the analysis failed to show a predictive role of treatment-dependent IMT changes.
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Influence of propofol on neuronal damage and apoptotic factors after incomplete cerebral ischemia and reperfusion in rats: a long-term observation.

TL;DR: These data show sustained neuroprotection with propofol related to reduced eosinophilic and apoptotic injury, and suggests the presence of activated caspase-3–independent apoptotic pathways.