R
Ricardo A. Machon
Researcher at Loyola Marymount University
Publications - 17
Citations - 1714
Ricardo A. Machon is an academic researcher from Loyola Marymount University. The author has contributed to research in topics: Schizophrenia (object-oriented programming) & Schizophrenia. The author has an hindex of 11, co-authored 17 publications receiving 1637 citations. Previous affiliations of Ricardo A. Machon include University of Southern California.
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Adult Schizophrenia Following Prenatal Exposure to an Influenza Epidemic
TL;DR: It is suggested that it is less the type than the timing of the disturbance during fetal neural development that is critical in determining risk for schizophrenia.
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Adult Major Affective Disorder After Prenatal Exposure to an Influenza Epidemic
TL;DR: Data from a population exposed during the second trimester to the 1957 influenza epidemic are consistent with the hypothesis concerning the possible neurodevelopmental contribution to the origins of some forms of major affective disorder, especially unipolar depressive disorder.
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The interaction of seasonality, place of birth, genetic risk and subsequent schizophrenia in a high risk sample.
TL;DR: The viral hypothesis that genetically vulnerable individuals, born in winter, in an urban environment would be more likely, as foetuses or neonates, to have suffered some CNS damage due to the infection, would show higher rates of schizophrenia diagnoses was supported.
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Hospitalization patterns in schizophrenia: A 13-year follow-up
TL;DR: All first admitted patients in 1972 from a catchment area of 582,000 inhabitants aged 15 years or more who were diagnosed as schizophrenic at least once from 1972 until September 1983 were followed-up on average 13 years after first admission.
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Prenatal factors in the pathogenesis of schizophrenia.
TL;DR: The observed association between prenatal viral infection and increased incidence of adult schizophrenia need not be a direct cytotoxic result of the viral infection, but may be caused indirectly from foetal minor cerebral haemorrhages produced by the anticoagulant effects of aspirin.