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Richard G. Taylor
Researcher at University of Arizona
Publications - 14
Citations - 2137
Richard G. Taylor is an academic researcher from University of Arizona. The author has contributed to research in topics: Calpain & Calpastatin. The author has an hindex of 11, co-authored 11 publications receiving 2066 citations. Previous affiliations of Richard G. Taylor include University of California, San Francisco.
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Is Z-disk degradation responsible for postmortem tenderization?
TL;DR: Electron microscope studies described in this paper show that, during the first 3 or 4 d of postmortem storage at 4 degrees C, both costameres and N2 lines are degraded.
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Hepatocyte growth factor activates quiescent skeletal muscle satellite cells in vitro
TL;DR: It is shown that HGF could act as an activator of quiescent satellite cells in vivo and the ability of HGF to stimulate precocious entry into the cell cycle is indicated.
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Role of the calpain system in muscle growth
TL;DR: Activity of the calpains in living cells is regulated by calpastatin and Ca2+, but the nature of this regulation is still unclear.
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Is calpain activity regulated by membranes and autolysis or by calcium and calpastatin
TL;DR: Although the Ca2+‐dependent proteinase (calpain) system has been found in every vertebrate cell that has been examined for its presence and has been detected in Drosophila and parasites, the physiological function(s) of this system remains unclear.
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Evidence that activation of platelet calpain is induced as a consequence of binding of adhesive ligand to the integrin, glycoprotein IIb-IIIa.
TL;DR: It is shown that calpain rapidly moved to a peripheral location upon the addition of an agonist to suspensions of platelets, but it remained unactivated, and binding of the extracellular domain of GP IIb-IIIa to its adhesive ligand can initiate a transmembrane signal that activates intracellular calpain.