R
Ritesh K Shukla
Researcher at Ahmedabad University
Publications - 48
Citations - 2256
Ritesh K Shukla is an academic researcher from Ahmedabad University. The author has contributed to research in topics: Comet assay & DNA damage. The author has an hindex of 13, co-authored 43 publications receiving 1914 citations. Previous affiliations of Ritesh K Shukla include Indian Institute of Toxicology Research & National Institute for Interdisciplinary Science and Technology.
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DNA damaging potential of zinc oxide nanoparticles in human epidermal cells.
TL;DR: The data demonstrates that ZnO NPs even at low concentrations possess a genotoxic potential in human epidermal cells which may be mediated through lipid peroxidation and oxidative stress, Hence, caution should be taken in their use in dermatological preparations as well as while handling.
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ROS-mediated genotoxicity induced by titanium dioxide nanoparticles in human epidermal cells
TL;DR: It is demonstrated that TiO(2) NPs have a mild cytotoxic potential, however, they induce ROS and oxidative stress leading to oxidative DNA damage and micronucleus formation, a probable mechanism of genotoxicity.
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TiO(2) nanoparticles induce oxidative DNA damage and apoptosis in human liver cells.
TL;DR: This study systematically shows that TiO2 NPs induce DNA damage and cause apoptosis in HepG2 cells even at very low concentrations, and the use of such nanoparticles should be carefully monitored.
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Surface functionalization of quantum dots for biological applications.
TL;DR: This review addresses the recent research progress made to improve aqueous solubility, functionalization of biomolecules to QD surface and the poorly understood chemistry involved in the steps of bio-functionalization of such nanoparticles.
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Toxicity of graphene in normal human lung cells (BEAS-2B).
N.V. Srikanth Vallabani,Sandeep Mittal,Ritesh K Shukla,Alok K. Pandey,Sanjay R. Dhakate,Renu Pasricha,Alok Dhawan +6 more
TL;DR: This study characrerized the GO using dynamic light scattering (DLS) along with the toxicological aspects related to cytotoxicity and apoptosis in normal human lung cells (BEAS-2B).