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Robert A. Wiswell

Researcher at University of Southern California

Publications -  57
Citations -  3154

Robert A. Wiswell is an academic researcher from University of Southern California. The author has contributed to research in topics: VO2 max & Lean body mass. The author has an hindex of 25, co-authored 57 publications receiving 3009 citations.

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Journal ArticleDOI

Muscle hypertrophy response to resistance training in older women

TL;DR: It is concluded that a program of resistance exercise can be safely carried out by elderly women, such a program significantly increases muscle strength, and such gains are due, at least in part, to muscle hypertrophy.
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Rate and mechanism of maximal oxygen consumption decline with aging: implications for exercise training.

TL;DR: Current evidence supports a 10% per decade decline in V̇O2max in men and women regardless of activity level, and aging most likely plays a role as studies have demonstrated that training maintenance becomes more difficult with advancing age.
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Relationship of body composition, muscle strength, and aerobic capacity to bone mineral density in older men and women.

TL;DR: It is concluded that body mass and grip strength, but not aerobic capacity, significantly predict bone density in elderly women, and VO2max per kilogram lean mass and back strength both make significant contributions to the prediction of spine mineral density.
Journal ArticleDOI

Muscle Hypertrophy Response To Resistance Training in Older Women

TL;DR: It is concluded that a program of resistance exercise can be safely carried out by elderly women, such a program significantly increases muscle strength, and such gains are due, at least in part, to muscle hypertrophy.
Journal ArticleDOI

Satellite cell numbers in young and older men 24 hours after eccentric exercise

TL;DR: It is concluded that a single bout of maximal eccentric exercise increases satellite cell numbers in both age groups, with a significantly greater response among the young men, and age‐related changes in satellite cell recruitment may contribute to muscle regeneration deficits among the elderly.