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Robert J. Chabot

Researcher at New York University

Publications -  33
Citations -  2315

Robert J. Chabot is an academic researcher from New York University. The author has contributed to research in topics: Population & Traumatic brain injury. The author has an hindex of 18, co-authored 33 publications receiving 2187 citations.

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Quantitative electroencephalographic profiles of children with attention deficit disorder

TL;DR: Results indicate neurophysiological dysfunction within the cortical and subcortical structures that serve the frontal/striatal system and models suggesting both hypo- or hyperarousal of these structures are supported.
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Quantitative EEG changes associated with loss and return of consciousness in healthy adult volunteers anaesthetized with propofol or sevoflurane

TL;DR: Alterations in the level of arousal were accompanied by significant QEEG changes, many of which were consistent across anaesthetic protocols, and frontal power predominance increased with deeper sedation, involving alpha and, to a lesser extent, delta and theta power.
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Sensitivity and specificity of QEEG in children with attention deficit or specific developmental learning disorders.

TL;DR: This paper provides a replication of all presented discriminant functions, and should provide the research basis for the generalized utilization of QEEG in the initial evaluation of children with learning and/or attention disorders.
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Acute effects and recovery after sport-related concussion: a neurocognitive and quantitative brain electrical activity study.

TL;DR: QEEG results suggest that the duration of physiological recovery after concussion may extend longer than observed clinical recovery, and QEEG studies revealed significant abnormalities in electrical brain activity in the injured group on day of injury and day 8 postinjury, but not on day 45.
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Patient State Index: titration of delivery and recovery from propofol, alfentanil, and nitrous oxide anesthesia.

TL;DR: Patient State Index–directed titration of prop ofol delivery resulted in faster emergence and recovery from propofol–alfentanil–nitrous oxide anesthesia, with modest decrease in the amount of propOfol delivered, without increasing the number of unwanted events.