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Robert J. Hamm

Researcher at Virginia Commonwealth University

Publications -  111
Citations -  8181

Robert J. Hamm is an academic researcher from Virginia Commonwealth University. The author has contributed to research in topics: Traumatic brain injury & Morris water navigation task. The author has an hindex of 47, co-authored 111 publications receiving 7910 citations. Previous affiliations of Robert J. Hamm include VCU Medical Center.

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A fluid percussion model of experimental brain injury in the rat

C. E. Dixon, +7 more
- 01 Jul 1987 - 
TL;DR: The data demonstrate that fluid percussion injury in the rat reproduces many of the features of head injury observed in other models and species, and could represent a useful experimental approach to studies of pathological changes similar to those seen in human head injury.
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The rotarod test: an evaluation of its effectiveness in assessing motor deficits following traumatic brain injury.

Robert J. Hamm, +4 more
- 01 Jan 1994 - 
TL;DR: The findings suggest that compared to the beam-balance and beam-walking tasks, the rotarod task is a more sensitive and efficient index for assessing motor impairment produced by brain injury.
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Marked Protection by Moderate Hypothermia after Experimental Traumatic Brain Injury

Guy L. Clifton, +5 more
- 01 Jan 1991 - 
TL;DR: Hypothermia-treated rats had significantly less beam-walking beam-balance, and body weight loss deficits compared to normothermic (38°C) rats, and the greatest protection was observed in the 30°C hypothermia group.
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Prolonged memory impairment in the absence of hippocampal cell death following traumatic brain injury in the rat.

Bruce G. Lyeth, +7 more
- 03 Sep 1990 - 
TL;DR: Mild to moderate TBI is capable of producing prolonged spatial memory deficits in the rat without evidence of either neuronal cell death in the intrinsic hippocampus or overt axonal injury in hippocampal pathways.
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Cognitive deficits following traumatic brain injury produced by controlled cortical impact.

Robert J. Hamm, +6 more
- 01 Jan 1992 - 
TL;DR: The results of the present experiment support the hypothesis that the hippocampus is preferentially vulnerable to damage following traumatic brain injury, and demonstrate that controlled cortical impact brain injury produces enduring cognitive deficits analogous to those observed after human brain injury.