Robert V. Gallo

TL;DR: Pulsatile secretion of LH, in the rat at least, does not appear to be regulated sloely by the medial basal hypothalamus, and central noradrenergic, cholinergic, dopaminergic, and serotoninergic systems are involved in influencing episodic LH release.

TL;DR: It is indicated that pulsatile LH release occurs during the ovulatory LII surge on proestrus in the rat, and that the rise, plateau, and fall in blood LH levels can be accounted for by changes in the LH pulse amplitude and frequency during different phases of the surge.

TL;DR: LH release is pulsatile during periods of low level LH secretion in the rat estrous cycle, and changes in the pulsatile characteristics of this release occur with different stages of the cycle.

TL;DR: In adult ovariectomized rats norepinephrine may be an excitatory neurotransmitter in the modulation of episodic LH release, and the activation of dopamine receptors may be capable of inhibiting this release process.

TL;DR: This study examined alterations in episodic LH release in response to prolonged, slow infusions of dopamine, norepinephrine, or epinephrine into the thire ventricle in adult, ovariectomized (OVX) rats, and the influence of priming with ovarian steroids on the LH response to the catecholamines.