R
Roland R. Roy
Researcher at University of California, Los Angeles
Publications - 483
Citations - 33896
Roland R. Roy is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Spinal cord & Spinal cord injury. The author has an hindex of 94, co-authored 440 publications receiving 31876 citations. Previous affiliations of Roland R. Roy include University of Washington & University of California, San Diego.
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Journal ArticleDOI
Muscle Architecture of the Human Lower Limb
TL;DR: The architectural features of the major knee extensors and flexors and ankle plantarflexors and dorsiflexors were determined in three human cadavers and grouped according to whether they showed a predilection for tension or velocity of shortening.
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Electrophoretic separation of rat skeletal muscle myosin heavy-chain isoforms
Robert J. Talmadge,Roland R. Roy +1 more
TL;DR: The percent composition of the four adult MHCs in rat soleus, medial gastrocnemius, diaphragm, and levator ani muscles by use of this procedure and Coomasie Blue staining is similar to that previously reported.
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Voluntary Exercise Induces a BDNF-Mediated Mechanism That Promotes Neuroplasticity
TL;DR: Investigation of potential mechanisms by which exercise can promote changes in neuronal plasticity via modulation of neurotrophins indicates that basal levels of neuromuscular activity are required to maintain normal levels of BDNF in the neuromUScular system and the potential for neuroplasticity.
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Recovery of supraspinal control of stepping via indirect propriospinal relay connections after spinal cord injury.
Grégoire Courtine,Bingbing Song,Roland R. Roy,Hui Zhong,Julia E. Herrmann,Yan Ao,Jingwei Qi,V. Reggie Edgerton,Michael V. Sofroniew +8 more
TL;DR: It is shown that pronounced functional recovery can occur after severe SCI without the maintenance or regeneration of direct projections from the brain past the lesion and can be mediated by the reorganization of descending and propriospinal connections.
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Transformation of nonfunctional spinal circuits into functional states after the loss of brain input.
Grégoire Courtine,Yury Gerasimenko,Rubia van den Brand,Aileen Yew,Pavel Musienko,Hui Zhong,Bingbing Song,Yan Ao,Ronaldo M. Ichiyama,Igor Lavrov,Roland R. Roy,Michael V. Sofroniew,V. Reggie Edgerton +12 more
TL;DR: It is proposed that, in the absence of supraspinal input, spinal locomotion can emerge from a combination of central pattern-generating capability and the ability of these spinal circuits to use sensory afferent input to control stepping.