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Rupert J. Quinnell
Researcher at University of Leeds
Publications - 106
Citations - 6486
Rupert J. Quinnell is an academic researcher from University of Leeds. The author has contributed to research in topics: Population & Necator americanus. The author has an hindex of 43, co-authored 99 publications receiving 6027 citations. Previous affiliations of Rupert J. Quinnell include University of London & University of Oxford.
Papers
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Journal ArticleDOI
Heterogeneities in the transmission of infectious agents: implications for the design of control programs.
Mark E. J. Woolhouse,Christopher Dye,J.-F. Etard,Thomas J. Smith,J.D. Charlwood,G. P. Garnett,Paul Hagan,J. L. K. Hii,Patricia D. Ndhlovu,Rupert J. Quinnell,Charlotte Watts,S. K. Chandiwana,Roy M. Anderson +12 more
TL;DR: From an analysis of the distributions of measures of transmission rates among hosts, an empirical relationship is identified suggesting that, typically, 20% of the host population contributes at least 80%" of the net transmission potential, as measured by the basic reproduction number, R0.
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Transmission, reservoir hosts and control of zoonotic visceral leishmaniasis
TL;DR: A review of the transmission and control of ZVL can be found in this article, which highlights the lack of randomized controlled trials of both dog culling and residual insecticide spraying.
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Infectiousness in a Cohort of Brazilian Dogs: Why Culling Fails to Control Visceral Leishmaniasis in Areas of High Transmission
Orin Courtenay,Orin Courtenay,Rupert J. Quinnell,Rupert J. Quinnell,Lourdes Maria Garcez,Jeffrey Jon Shaw,Christopher Dye +6 more
TL;DR: Mathematical modeling suggests that culling programs fail because of high incidence of infection and infectiousness, the insensitivity of the diagnostic test to detect infectious dogs, and time delays between diagnosis and culling.
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Do helminth parasites protect against atopy and allergic disease
TL;DR: Current cross‐sectional, birth cohort, and intervention study evidence for a protective effect of helminth infection on allergy is reviewed and a genetically predetermined T‐helper type 2 cell‐dominated cytokine milieu reduces parasite burden and may enhance host survival in an environment where helmineth parasites are prevalent.
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Genetics of susceptibility to human helminth infection.
TL;DR: Human genome scans have identified a locus responsible for controlling Schistosoma mansoni infection intensity on chromosome 5q31-q33, and loci controlling Ascaris lumbricoides intensity on chromosomes 1 and 13, although the genes involved have not yet been identified.