R
Russell D. Cox
Researcher at Icahn School of Medicine at Mount Sinai
Publications - 7
Citations - 370
Russell D. Cox is an academic researcher from Icahn School of Medicine at Mount Sinai. The author has contributed to research in topics: Hippocampal formation & Serotonergic cell groups. The author has an hindex of 7, co-authored 7 publications receiving 368 citations.
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Journal ArticleDOI
Nocturnal rotation in normal rats: Correlation with amphetamine-induced rotation and effects of nigro-striatal lesions
Stanley D. Glick,Russell D. Cox +1 more
TL;DR: It is suggested that, following a unilateral lesion, compensatory processes occur to a greater extent if the lesion is in the normally more active side of the brain.
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Differential effects of unilateral and bilateral caudate lesions on side preferences and timing behavior in rats.
Stanley D. Glick,Russell D. Cox +1 more
TL;DR: It is suggested that side preferences are intimately involved in the control of behavior by internal stimuli and that an inherent asymmetry in nigrostriatal function underlies side preferences; the effect of a unilateral striatal lesion will depend upon whether the lesion is placed in the more or less active striatum.
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Relationship of rats' spatial preferences effects of d-amphetamine on timing behavior
TL;DR: Observations of bar-pressing behavior suggested that stereotyped motor patterns associated with side preferences might be related to mechanisms involved in timing behavior and perhaps, in behavior controlled by internal stimuli generally.
Journal ArticleDOI
Changes in morphine self-administration after tel-diencephalic lesions in rats.
Stanley D. Glick,Russell D. Cox +1 more
TL;DR: Doseresponse studies indicated that sensitivity to morphine's rewarding property was decreased by frontal cortical and hippocampal lesions, and a neuroanatomical substrate for morphine reinforcement is suggested.
Journal ArticleDOI
Changes in morphine self-administration after brainstem lesions in rats.
Stanley D. Glick,Russell D. Cox +1 more
TL;DR: Dose-response studies indicated that sensitivity to morphine's rewarding property was increased by substantia nigra lesions and decreased by medial raphe lesions, which appears to be involved in the mechanism of morphine reinforcement.