scispace - formally typeset
R

Ruth A. Carper

Researcher at San Diego State University

Publications -  34
Citations -  6242

Ruth A. Carper is an academic researcher from San Diego State University. The author has contributed to research in topics: Autism & Frontal lobe. The author has an hindex of 16, co-authored 32 publications receiving 5787 citations. Previous affiliations of Ruth A. Carper include University of California, San Diego & Boston Children's Hospital.

Papers
More filters
Journal ArticleDOI

Unusual brain growth patterns in early life in patients with autistic disorder: An MRI study

TL;DR: Hyperplasia was present in cerebral gray matter and cerebral and cerebellar white matter in early life in patients with autism and normal regulation of brain growth in autism results in early overgrowth followed by abnormally slowed growth.
Journal ArticleDOI

Autism and abnormal development of brain connectivity

TL;DR: It has been said that people with autism suffer from a lack of “central coherence,” the cognitive ability to bind together a jumble of separate features into a single, coherent object or concept.
Journal ArticleDOI

Evidence of brain overgrowth in the first year of life in autism.

TL;DR: The clinical onset of autism appears to be preceded by 2 phases of brain growth abnormality: a reduced head size at birth and a sudden and excessive increase in head size between 1 to 2 months and 6 to 14 months, which may serve as an early warning signal for autism.
Journal ArticleDOI

Cerebral Lobes in Autism: Early Hyperplasia and Abnormal Age Effects

TL;DR: It is found that frontal, temporal, and parietal white Matter volumes, as well as frontal and temporal gray matter volumes, changed at significantly slower rates in autism patients than in controls across the 2- to 11-year-age range.
Journal ArticleDOI

Longitudinal Magnetic Resonance Imaging Study of Cortical Development through Early Childhood in Autism

TL;DR: It is found that overgrowth clearly begins before 2 years of age, and future longitudinal studies would benefit from inclusion of even younger populations as well as further characterization of genetic and other biomarkers to determine the underlying neuropathological processes causing the onset of autistic symptoms.