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Ruth A. Hackett

Researcher at University College London

Publications -  40
Citations -  1523

Ruth A. Hackett is an academic researcher from University College London. The author has contributed to research in topics: Longitudinal study & Type 2 diabetes. The author has an hindex of 15, co-authored 36 publications receiving 969 citations. Previous affiliations of Ruth A. Hackett include King's College London.

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Type 2 diabetes mellitus and psychological stress - a modifiable risk factor.

TL;DR: The physiological responses to stress that are probably related to type 2 diabetes mellitus are highlighted and intervention studies that have attempted to modify psychological or social factors to improve outcomes in people with T2DM are given.
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Loneliness and stress-related inflammatory and neuroendocrine responses in older men and women.

TL;DR: Cortisol responsivity was inversely related to loneliness in women, with the odds of being a cortisol responder decreasing with increased loneliness independently of covariates, and the impact of loneliness on health in women may be mediated in part through dysregulation of inflammatory and neuroendocrine systems.
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Disruption of multisystem responses to stress in type 2 diabetes: Investigating the dynamics of allostatic load

TL;DR: The notion that people with type 2 diabetes experience chronic allostatic load, manifest as dynamic disturbances in reactivity to and recovery from stress across multiple biological systems, coupled with heightened experience of chronic life stress, provides a unifying perspective with implications for etiology and patient management.
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Diurnal Cortisol Patterns, Future Diabetes, and Impaired Glucose Metabolism in the Whitehall II Cohort Study

TL;DR: In this nonclinical population, alterations in diurnal cortisol patterns were predictive of future glucose disturbance in this community-dwelling population.
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Association of diurnal patterns in salivary cortisol with type 2 diabetes in the Whitehall II study.

TL;DR: In this nonclinical population, T2D was associated with a flatter slope in cortisol levels across the day and raised bedtime cortisol values and there was no association between morning cortisol, the cortisol awakening response, and T1D.