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Sarah Finn

Researcher at University College Dublin

Publications -  9
Citations -  535

Sarah Finn is an academic researcher from University College Dublin. The author has contributed to research in topics: Salmonella enterica & Salmonella. The author has an hindex of 8, co-authored 9 publications receiving 438 citations.

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Journal ArticleDOI

Mechanisms of survival, responses and sources of Salmonella in low-moisture environments.

TL;DR: This review summarizes the current state of knowledge with respect to Salmonella survival in intermediate- and low-moisture food matrices and their production environments and explores sources and potential control measures to reduce the risk of contamination.
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Elucidation of the RamA regulon in Klebsiella pneumoniae reveals a role in LPS regulation.

TL;DR: It is demonstrated that RamA-mediated regulation directly perturbs microbial surface properties, including lipid A biosynthesis, which facilitate evasion from the innate host response, highlighting RamA as a global regulator that confers pathoadaptive phenotypes with implications for the understanding of the pathogenesis of Enterobacter, Salmonella and Citrobacter spp.
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ProP Is Required for the Survival of Desiccated Salmonella enterica Serovar Typhimurium Cells on a Stainless Steel Surface

TL;DR: Knowing the responses of S. Typhimurium ST4/74 at the transcriptional level to desiccation on a stainless steel surface and to subsequent rehydration should prove valuable for understanding the underlying mechanisms involved in pathogen survival and should lead to improved methods for control to ensure the safety of intermediate- and low-moisture foods.
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Elucidating the Regulon of Multidrug Resistance Regulator RarA in Klebsiella pneumoniae

TL;DR: The transcriptome and phenotypic microarray data support and extend the role of RarA in the MDR phenotype of K. pneumoniae and demonstrate enhanced growth of the overexpresser in the presence of several antibiotic classes.
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Comparative analysis of Salmonella susceptibility and tolerance to the biocide chlorhexidine identifies a complex cellular defense network

TL;DR: Results indicated that chlorhexidine tolerance was associated with more extensive modifications of the same cellular processes involved in this proposed network, as well as a divergent defense response involving the up-regulation of additional targets and an altered cellular phosphate metabolism.