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Satvinder S. Dhaliwal

Researcher at Curtin University

Publications -  264
Citations -  7959

Satvinder S. Dhaliwal is an academic researcher from Curtin University. The author has contributed to research in topics: Chemistry & Medicine. The author has an hindex of 44, co-authored 210 publications receiving 6839 citations. Previous affiliations of Satvinder S. Dhaliwal include University of Western Australia & Diabetes Australia.

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Effects of calcium supplementation on clinical fracture and bone structure: Results of a 5-year, double-blind, placebo-controlled trial in elderly women

TL;DR: In this paper, the authors examined whether calcium supplementation decreases clinical fracture risk in elderly women and its mechanism of action and concluded that supplementing with calcium carbonate tablets supplying 1200 mg/d is ineffective as a public health intervention in preventing clinical fractures in the ambulatory elderly population owing to poor long-term compliance, but it is effective in those patients who are compliant.
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Waist–hip ratio is the dominant risk factor predicting cardiovascular death in Australia

TL;DR: Clinical measures of obesity are evaluated for their ability to predict death from cardiovascular disease (CVD) and coronary heart disease (CHD) in parallel with conventional cardiovascular risk factors.
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The effect of 12 weeks of aerobic, resistance or combination exercise training on cardiovascular risk factors in the overweight and obese in a randomized trial

TL;DR: A 12-week training program comprising of resistance or combination exercise, at moderate-intensity for 30 min, five days/week resulted in improvements in the cardiovascular risk profile in overweight and obese participants compared to no exercise.
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Effects of whey protein isolate on body composition, lipids, insulin and glucose in overweight and obese individuals

TL;DR: It is demonstrated that supplementation with whey proteins improves fasting lipids and insulin levels in overweight and obese individuals.
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Blood-brain barrier dysfunction developed during normal aging is associated with inflammation and loss of tight junctions but not with leukocyte recruitment.

TL;DR: The BBB breakdown that occurs during ordinary aging is associated with inflammation and disruption of tight junction complex assembly but not through leukocyte trafficking.