scispace - formally typeset
S

Shao-wei Li

Researcher at Wenzhou Medical College

Publications -  28
Citations -  204

Shao-wei Li is an academic researcher from Wenzhou Medical College. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 2, co-authored 15 publications receiving 23 citations.

Papers
More filters
Journal ArticleDOI

Effect of Hepatic Macrophage Polarization and Apoptosis on Liver Ischemia and Reperfusion Injury During Liver Transplantation.

TL;DR: The effects of hepatic macrophage polarization and apoptosis on liver I/R are summarized and a new notion has been proposed that KC apoptosis may influence I-R in yet another manner as well.
Journal ArticleDOI

Reactive Oxygen Species Induce Fatty Liver and Ischemia-Reperfusion Injury by Promoting Inflammation and Cell Death

TL;DR: The effects of reactive oxygen species on ischemia-reperfusion injury and non-alcoholic fatty liver injury are reviewed as well as several treatment approaches are highlighted.
Journal ArticleDOI

Animal and Organoid Models of Liver Fibrosis.

TL;DR: In this article, the authors focused on the modeling methods and fibrosis characteristics of different animal models of liver fibrosis, such as a chemical-induced liver-fibrosis model, autoimmune liver-influenced liver-deficiency disease (LDF), cholestatic liver-fluoroacetylcholine (Choline) model, alcoholic liver-foreign membrane protein (ALF) model and non-alcoholic liver-fluroacetylaclavase (LFA) model.
Journal ArticleDOI

Liver injury in COVID-19: Detection, pathogenesis, and treatment.

TL;DR: In this paper, the authors present an analysis of the clinical features, potential mechanisms, and treatment strategies for liver injury associated with COVID-19, and hope that this review would benefit clinicians in devising better strategies for management of such patients.
Journal ArticleDOI

Novel Targets and Therapeutic Strategies to Protect Against Hepatic Ischemia Reperfusion Injury

TL;DR: An overview of the latest advances of treatment strategies and proposed potential mechanisms behind liver IRI is presented and the role of several important molecules (PPARγ, FAM3A, and non-coding RNAs) in protecting against hepatic IRI are highlighted.