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Shao-wei Li
Researcher at Wenzhou Medical College
Publications - 28
Citations - 204
Shao-wei Li is an academic researcher from Wenzhou Medical College. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 2, co-authored 15 publications receiving 23 citations.
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Journal ArticleDOI
Effect of Hepatic Macrophage Polarization and Apoptosis on Liver Ischemia and Reperfusion Injury During Liver Transplantation.
TL;DR: The effects of hepatic macrophage polarization and apoptosis on liver I/R are summarized and a new notion has been proposed that KC apoptosis may influence I-R in yet another manner as well.
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Reactive Oxygen Species Induce Fatty Liver and Ischemia-Reperfusion Injury by Promoting Inflammation and Cell Death
TL;DR: The effects of reactive oxygen species on ischemia-reperfusion injury and non-alcoholic fatty liver injury are reviewed as well as several treatment approaches are highlighted.
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Animal and Organoid Models of Liver Fibrosis.
Yu-long Bao,Li Wang,Hai-ting Pan,Tai-ran Zhang,Ya-Hong Chen,Shan-jing Xu,Xin-Li Mao,Xin-Li Mao,Shao-wei Li +8 more
TL;DR: In this article, the authors focused on the modeling methods and fibrosis characteristics of different animal models of liver fibrosis, such as a chemical-induced liver-fibrosis model, autoimmune liver-influenced liver-deficiency disease (LDF), cholestatic liver-fluoroacetylcholine (Choline) model, alcoholic liver-foreign membrane protein (ALF) model and non-alcoholic liver-fluroacetylaclavase (LFA) model.
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Liver injury in COVID-19: Detection, pathogenesis, and treatment.
TL;DR: In this paper, the authors present an analysis of the clinical features, potential mechanisms, and treatment strategies for liver injury associated with COVID-19, and hope that this review would benefit clinicians in devising better strategies for management of such patients.
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Novel Targets and Therapeutic Strategies to Protect Against Hepatic Ischemia Reperfusion Injury
TL;DR: An overview of the latest advances of treatment strategies and proposed potential mechanisms behind liver IRI is presented and the role of several important molecules (PPARγ, FAM3A, and non-coding RNAs) in protecting against hepatic IRI are highlighted.