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Sheri J. Y. Mizumori

Researcher at University of Washington

Publications -  107
Citations -  6761

Sheri J. Y. Mizumori is an academic researcher from University of Washington. The author has contributed to research in topics: Hippocampal formation & Hippocampus. The author has an hindex of 45, co-authored 103 publications receiving 6274 citations. Previous affiliations of Sheri J. Y. Mizumori include University of Colorado Boulder & University of Utah.

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Disruption of NMDAR-dependent burst firing by dopamine neurons provides selective assessment of phasic dopamine-dependent behavior.

TL;DR: The role of phasic DA was addressed by attenuating DA neuron burst firing and subsequent DA release, without altering tonic neural activity, and dramatically attenuated learning about cues that predicted rewarding and aversive events while leaving many other DA-dependent behaviors unaffected.
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Directionally selective mnemonic properties of neurons in the lateral dorsal nucleus of the thalamus of rats

TL;DR: It was concluded that the LDN may pass on to the hippocampal formation directional information that is not merely a reflection of current sensory input, and may serve an important integrative function for limbic spatial learning systems.
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Hippocampal place cells, context, and episodic memory

TL;DR: Recent data from the laboratory, together with previous findings, indicate that hippocampal place fields and neuronal responses to task‐relevant stimuli are highly sensitive to the context, even when the contexts are defined by abstract task demands rather than the spatial geometry of the environment.
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Role of the dorsomedial striatum in behavioral flexibility for response and visual cue discrimination learning.

TL;DR: Findings contrast with findings indicating that prelimbic inactivation impairs behavioral flexibility due to perseveration of a previously learned strategy, but each region may contribute to distinct processes that facilitate strategy switching.
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Activation of dopamine neurons is critical for aversive conditioning and prevention of generalized anxiety

TL;DR: A mouse model in which activation of dopamine neurons in response to an aversive stimulus is attenuated by conditional genetic inactivation of functional NMDA receptors on dopamine neurons is described, which is consistent with a role for dopamine in facilitating contingency awareness that is critical for the prevention of generalized anxiety.