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Shilpa Rao

Researcher at National Institute of Mental Health and Neurosciences

Publications -  62
Citations -  238

Shilpa Rao is an academic researcher from National Institute of Mental Health and Neurosciences. The author has contributed to research in topics: Medicine & Glioma. The author has an hindex of 7, co-authored 45 publications receiving 140 citations.

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LIN28A, a sensitive immunohistochemical marker for Embryonal Tumor with Multilayered Rosettes (ETMR), is also positive in a subset of Atypical Teratoid/Rhabdoid Tumor (AT/RT)

TL;DR: Among CNS embryonal tumors, LIN28A is not specific to ETMRs and such immunoreactivity can also be seen in a proportion of AT/RTs, and this study reiterates that LIN 28A is a sensitive IHC marker for the diagnosis of ETMR.
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Differential gene expression in peritumoral brain zone of glioblastoma: role of SERPINA3 in promoting invasion, stemness and radioresistance of glioma cells and association with poor patient prognosis and recurrence

TL;DR: Serine protease inhibitor clade A, member 3 (SERPINA3) was selected for further functional characterization in vitro by gene knockdown approach in glioma cells and showed decreased tumor cell proliferation, invasion, migration, transition to mesenchymal phenotype, stemness and radioresistance.
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Afferent pathways of sympathetic skin response in spinal cord: a clinical and electrophysiological study

TL;DR: Spinal afferents of SSR are closely related with tracts of bladder sensations and are different from pathways for SEP, according to standard neurological classification of spinal cord injury.
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A simple algorithmic approach using histology and immunohistochemistry for the current classification of adult diffuse glioma in a resource-limited set-up.

TL;DR: In a resource-limited set-up, histology with IHC (IDH1(R132H) and ATRX) form the baseline to reasonably derive four histomolecular subgroups of diffuse glioma.
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Granulomatous and lymphocytic hypophysitis - are they immunologically distinct?

TL;DR: GH revealed cytotoxic T cell and histiocyte rich infiltrate in contrast to CD4+ T‐cell predominance in LH suggesting that the two forms have distinct immunological mechanisms in evolution, an autoimmune process in LH and type IV hypersensitivity response in GH.