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Shou-Dong Lee

Researcher at National Yang-Ming University

Publications -  790
Citations -  27551

Shou-Dong Lee is an academic researcher from National Yang-Ming University. The author has contributed to research in topics: Cirrhosis & Portal hypertension. The author has an hindex of 75, co-authored 788 publications receiving 26066 citations. Previous affiliations of Shou-Dong Lee include Kowloon Hospital & National Defense Medical Center.

Papers
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Genetic Diversity of Hepatitis B Virus Strains Derived Worldwide: Genotypes, Subgenotypes, and HBsAg Subtypes

Helene Norder, +8 more
- 01 Nov 2004 - 
TL;DR: The genetic diversity ofHBV and the geographical distribution of its subgenotypes provide a tool to reconstruct the evolutionary history of HBV and may help to complement genetic data in the understanding of the evolution and past migrations of man.
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Des-gamma-carboxy (abnormal) prothrombin as a serum marker of primary hepatocellular carcinoma.

Howard A. Liebman, +7 more
- 31 May 1984 - 
TL;DR: Levels of the abnormal prothrombin were low in patients with chronic active hepatitis or metastatic carcinoma involving the liver, and undetectable in normal subjects, while Serum α-fetoprotein levels correlated poorly with abnormal-prothrom bin levels.
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Peginterferon alpha-2a (40 kDa): an advance in the treatment of hepatitis B e antigen-positive chronic hepatitis B.

W. G. E. Cooksley, +9 more
- 01 Jul 2003 - 
TL;DR: Peginterferon α‐2a (40 kDa) is superior to conventional interferonα‐2A in the treatment of chronic hepatitis C, and this is the first report on peginterferon β‐1a ( 40 kDa) in the Treatment of CHB.
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Polymorphism of the N‐acetyltransferase 2 gene as a susceptibility risk factor for antituberculosis drug–induced hepatitis

Yi-Shin Huang, +7 more
- 01 Apr 2002 - 
TL;DR: Slow‐acetylator status of NAT2 is a significant susceptibility risk factor for antituberculosis drug–induced hepatitis and slow acetylators are prone to develop more severe hepatotoxicity than rapid acetylator.
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Cytochrome P450 2E1 genotype and the susceptibility to antituberculosis drug-induced hepatitis.

Yi Shin Huang, +7 more
- 01 Apr 2003 - 
TL;DR: Under the administration of isoniazid, the volunteers with CYP 2E1 c1/c1 genotype had higher CYP2E1 activity than those with other genotypes had and, hence, might produce more hepatotoxins.