S
Simon Hannam
Researcher at King's College London
Publications - 65
Citations - 1626
Simon Hannam is an academic researcher from King's College London. The author has contributed to research in topics: Sudden infant death syndrome & Supine position. The author has an hindex of 21, co-authored 64 publications receiving 1502 citations. Previous affiliations of Simon Hannam include University of Cambridge.
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Journal ArticleDOI
The role of Toll-like receptors in renal diseases
TL;DR: Evidence supporting a role for TLRs in contrasting bacterial infections and in causing or aggravating renal conditions when TLR activation leads to a harmful inflammatory response is discussed.
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Effect of Prone and Supine Position on Sleep, Apneas, and Arousal in Preterm Infants
Ravindra Bhat,Simon Hannam,Ronit M. Pressler,Gerrard F. Rafferty,Janet L. Peacock,Anne Greenough +5 more
TL;DR: Very prematurely born infants studied before neonatal unit discharge sleep more efficiently with fewer arousals and more central apneas in the prone position, emphasizing the importance of recommending supine sleeping after neonatal units discharge for prematurely born babies.
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Neonatal abstinence syndrome--postnatal ward versus neonatal unit management.
TL;DR: Testing the hypothesis that caring for infants with neonatal abstinence syndrome with their mothers on the postnatal ward rather than admit them to the neonatal unit would reduce treatment duration and length of hospital stay suggests that this suggestion is correct.
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Takotsubo cardiomyopathy--the current state of knowledge.
Agata Bielecka-Dabrowa,Dimitri P. Mikhailidis,Simon Hannam,Jacek Rysz,Marta Michalska,Yoshihiro J. Akashi,Maciej Banach +6 more
TL;DR: The temporal relationship between the stressful event and the triggering of the clinical syndrome as well as the report of elevated catecholamine plasma levels during the acute phase suggest a possible involvement of the sympathetic nervous system.
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Metabolic syndrome and renal disease
TL;DR: There has been little evidence that preventing or treating symptoms of the MetS protects patients from renal impairment, and obesity may enhance the risk of renal dysfunction development probably through mechanisms associated with renal hyperfiltration, hyperperfusion and focal glomerulosclerosis.