S
Simon P. Frostick
Researcher at University of Liverpool
Publications - 130
Citations - 6974
Simon P. Frostick is an academic researcher from University of Liverpool. The author has contributed to research in topics: Tennis elbow & Electromyography. The author has an hindex of 38, co-authored 127 publications receiving 6574 citations. Previous affiliations of Simon P. Frostick include University of Nottingham & University of Oxford.
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Journal ArticleDOI
Dabigatran etexilate versus enoxaparin for prevention of venous thromboembolism after total hip replacement: a randomised, double-blind, non-inferiority trial
Bengt I. Eriksson,Ola E. Dahl,Nadia Rosencher,Andreas A. Kurth,C Niek van Dijk,Simon P. Frostick,Martin H. Prins,Rohan Hettiarachchi,Stefan Hantel,Janet Schnee,Harry R. Büller +10 more
TL;DR: Oral dabigatran etexilate was as effective as enoxaparin in reducing the risk of venous thromboembolism after total hip replacement surgery, with a similar safety profile.
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Oral dabigatran etexilate vs. subcutaneous enoxaparin for the prevention of venous thromboembolism after total knee replacement: the RE-MODEL randomized trial.
Bengt I. Eriksson,Ola E. Dahl,Nadia Rosencher,Andreas A. Kurth,C. N. Van Dijk,Simon P. Frostick,Peter Kälebo,Anita Vedel Christiansen,Stefan Hantel,Rohan Hettiarachchi,Janet Schnee,Harry R. Büller +11 more
TL;DR: Dabigatran etexilate (220 mg or 150‰mg) was at least as effective as enoxaparin and had a similar safety profile for prevention of VTE after total knee replacement surgery.
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Schwann cells, neurotrophic factors, and peripheral nerve regeneration
TL;DR: This review article focuses on the roles in peripheral nerve regeneration of Schwann cells and of the neurotrophin family, CNTF and GDNF, and the relationship between these and what remains to be understood about the possible clinical use of neurotrophic factors.
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Skeletal muscle metabolism in patients with congestive heart failure: relation to clinical severity and blood flow.
B M Massie,Michael A. Conway,R Yonge,Simon P. Frostick,J. G. G. Ledingham,Peter Sleight,George K. Radda,Bheeshma Rajagopalan +7 more
TL;DR: The results indicate that skeletal muscle metabolic abnormalities are present in many patients with CHF and that they are not primarily due to either muscle atrophy or impaired blood flow, which may explain in part the marked heterogeneity of symptom status and exercise capacity of patients with similar degrees of cardiac dysfunction.
Journal ArticleDOI
Skeletal muscle metabolism during exercise under ischemic conditions in congestive heart failure. Evidence for abnormalities unrelated to blood flow.
B M Massie,Michael A. Conway,Bheeshma Rajagopalan,R Yonge,Simon P. Frostick,J. G. G. Ledingham,Peter Sleight,G. K. Radda +7 more
TL;DR: The results indicate that in many patients with congestive heart failure exercising muscle exhibits increased glycolytic metabolism and appears to be metabolically less efficient in relation to external work performed, and cannot be explained by impaired blood flow or oxygen delivery alone.