S
Simone G. Shamay-Tsoory
Researcher at University of Haifa
Publications - 173
Citations - 12518
Simone G. Shamay-Tsoory is an academic researcher from University of Haifa. The author has contributed to research in topics: Empathy & Cognition. The author has an hindex of 49, co-authored 153 publications receiving 10550 citations. Previous affiliations of Simone G. Shamay-Tsoory include Ruhr University Bochum & Bar-Ilan University.
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Neurocognitive basis of impaired empathy in schizophrenia.
TL;DR: The degree of impairment in cognitive and affective empathy in schizophrenia is examined and the contribution of executive prefrontal functions to empathy in these patients is evaluated to explore the neurocognitive processes that underlie the empathic ability in schizophrenic patients.
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Herding Brains: A Core Neural Mechanism for Social Alignment
TL;DR: A model that views synchronized movement, emotional contagion, and social conformity as interrelated processes that rely on shared neural networks is proposed, suggesting that social alignment is mediated by a three-component feedback loop.
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Neuropsychological Evidence of Impaired Cognitive Empathy in Euthymic Bipolar Disorder
TL;DR: Impaired cognitive empathy was related with performance in neurocognitive tasks of cognitive flexibility, suggesting that prefrontal cortical dysfunction may account for impaired cognitive empathy in bipolar disorder.
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Oxytocin facilitates accurate perception of competition in men and kinship in women
TL;DR: It is concluded that the gender-specific findings reported here may point to some biosocial differences in the effect of OT which may be expressed in women's tendency for communal and familial social behavior as opposed to men's tendency to competitive social behavior.
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The neuroanatomical basis of affective mentalizing in schizophrenia: Comparison of patients with schizophrenia and patients with localized prefrontal lesions
TL;DR: The pattern of mentalizing impairments in schizophrenia resembled those seen in patients with lesions of the frontal lobe, particularly with VM damage, providing support for the notion of a disturbance of the fronto-limbic circuits in schizophrenia.