S
So-Young Lee
Researcher at Pohang University of Science and Technology
Publications - 10
Citations - 202
So-Young Lee is an academic researcher from Pohang University of Science and Technology. The author has contributed to research in topics: Trust management (information system) & hERG. The author has an hindex of 7, co-authored 10 publications receiving 188 citations.
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Journal ArticleDOI
Blockade of HERG human K+ channels and IKr of guinea-pig cardiomyocytes by the antipsychotic drug clozapine.
TL;DR: It is suggested that blockade of HERG currents and IKr, but not IKs, may contribute to the arrhythmogenic side effects of clozapine.
Journal ArticleDOI
Inhibition of Na+-K+ Pump and L-Type Ca2+ Channel by Glibenclamide in Guinea Pig Ventricular Myocytes
So-Young Lee,Chin Ok Lee +1 more
TL;DR: Results indicate that glibenclamide increases [Na+i] by inhibiting the Na+-K+ pump and decreases contraction and Ca2- transient, in addition, by blocking the L-type Ca2+ channel.
Proceedings ArticleDOI
A reputation management system in structured peer-to-peer networks
TL;DR: Simulation results show that the proposed reputation management system works better in preventing untrustworthy files from spreading than existing systems even in cases of allowing malicious peers to change their identities.
Journal ArticleDOI
Overexpression of junctate induces cardiac hypertrophy and arrhythmia via altered calcium handling.
Chang-Soo Hong,Soon-Jae Kwon,Myeong Chan Cho,Yong-Geun Kwak,Ki-Chan Ha,Bing-Zhe Hong,Haiying Li,Soo-Wan Chae,Ok Hee Chai,Chang Ho Song,Yuhua Li,Joon-Chul Kim,Sun-Hee Woo,So-Young Lee,Chin Ok Lee,Do Han Kim +15 more
TL;DR: It is shown that junctate-1 TG mice (8 weeks old) were under local PKA signaling that could cause increased L-type Ca2+ current and reduced SR function and these sequential changes could well be correlated with the physiological changes.
Journal ArticleDOI
Norepinephrine activates store-operated Ca2+ entry coupled to large-conductance Ca2+-activated K+ channels in rat pinealocytes.
TL;DR: It is reported that the Ca(2+) influx triggered by NE significantly regulates the protein level of serotonin N-acetyl transferase, or arylalkylamine N- acetyltransferase (AANAT), a critical enzyme in melatonin production, and is responsible for maintaining the Ca (2+) response after repetitive stimulation.