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Stefano Fumagalli
Researcher at University of Florence
Publications - 171
Citations - 4569
Stefano Fumagalli is an academic researcher from University of Florence. The author has contributed to research in topics: Medicine & Atrial fibrillation. The author has an hindex of 33, co-authored 149 publications receiving 3738 citations. Previous affiliations of Stefano Fumagalli include University of Strathclyde & University of Milan.
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Temporal pattern of expression and colocalization of microglia/macrophage phenotype markers following brain ischemic injury in mice
TL;DR: These data show that the ischemic lesion is accompanied by activation of specific microglia/macrophage phenotype that presents distinctive spatial and temporal features that provide a basis for understanding this complex response and for developing strategies resulting in promotion of a protective inflammatory phenotype.
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Improved exercise tolerance and quality of life with cardiac rehabilitation of older patients after myocardial infarction. Results of a randomized, controlled trial☆
Niccolò Marchionni,Francesco Fattirolli,Stefano Fumagalli,Neil Oldridge,Francesco Del Lungo,Linda Morosi,Costanza Burgisser,Giulio Masotti +7 more
TL;DR: Post-MI Hosp-CR and Home-CR are similarly effective in the short term and improve TWC and HRQL in each age group, but with lower costs and more prolonged positive effects, Home- CR may be the treatment of choice in low-risk older patients.
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Reduced Cardiocirculatory Complications With Unrestrictive Visiting Policy in an Intensive Care Unit Results From a Pilot, Randomized Trial
Stefano Fumagalli,Lorenzo Boncinelli,Antonella Lo Nostro,Paolo Valoti,Giorgio Baldereschi,Mauro Di Bari,Andrea Ungar,Samuele Baldasseroni,Pierangelo Geppetti,Giulio Masotti,Riccardo Pini,Niccolò Marchionni +11 more
TL;DR: Despite greater environmental microbial contamination, liberalizing visiting hours in ICUs does not increase septic complications, whereas it might reduce cardiovascular complications, possibly through reduced anxiety and more favorable hormonal profile.
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The ischemic environment drives microglia and macrophage function.
Stefano Fumagalli,Stefano Fumagalli,Carlo Perego,Francesca Pischiutta,Elisa R. Zanier,Maria Grazia De Simoni +5 more
TL;DR: The selective responses of microglia and macrophages to hypoxia after stroke are discussed and relevant markers are reviewed with the aim of defining the different subpopulations of myeloid cells that are recruited to the injured site.
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CX3CR1 deficiency induces an early protective inflammatory environment in ischemic mice
TL;DR: Data show that in cx3cr1−/− mice protection from ischemia at early time points after injury is associated with a protective inflammatory milieu, characterized by the promotion of M2 polarization markers.