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Susan J. Spencer
Researcher at University of California, San Francisco
Publications - 13
Citations - 757
Susan J. Spencer is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Adrenal gland & Fetus. The author has an hindex of 11, co-authored 13 publications receiving 746 citations.
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Proliferation and apoptosis in the human adrenal cortex during the fetal and perinatal periods: implications for growth and remodeling.
TL;DR: Growth of the human fetal adrenal cortex involves cellular hyperplasia, mainly in the DZ and to a lesser extent in the FZ, which is probably dependent on ACTH; and apoptosis occurs predominantly in the inner cortical compartment and may be responsible for the rapid regression of theFZ after birth, a process that may be regulated by activin A and/or TGFbeta.
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Activin and inhibin in the human adrenal gland. Regulation and differential effects in fetal and adult cells.
TL;DR: The data indicate that activin-A may be an autocrine or paracrine factor regulated by ACTH, involved in modulating growth and differentiated function of the human fetal adrenal gland.
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Recombinant human activin-A promotes proliferation of human luteinized preovulatory granulosa cells in vitro.
TL;DR: It is suggested that activin-A is able to modulate growth of ovarian granulosa cells in a dose-dependent manner.
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Activin-A as an intraovarian modulator: actions, localization, and regulation of the intact dimer in human ovarian cells.
TL;DR: Results indicate that activin-A may function as an autocrine or paracrine regulator of follicular function in the human ovary.
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Plasma apolipoprotein L concentrations correlate with plasma triglycerides and cholesterol levels in normolipidemic, hyperlipidemic, and diabetic subjects.
Philippe Duchateau,Irina Movsesyan,Shizuya Yamashita,Naohiko Sakai,Ken-ichi Hirano,Samantha A. Schoenhaus,Patricia M. O'Connor-Kearns,Susan J. Spencer,Robert B. Jaffe,Rita F. Redberg,Brian Y. Ishida,Yugi Matsuzawa,John P. Kane,Mary J. Malloy +13 more
TL;DR: Apolipoprotein L levels in plasma of patients with primary cholesteryl ester transfer protein deficiency significantly increased, and no significant correlation was found between apoL and body mass index, age, sex, HDL-cholesterol or fasting glucose and glycohemoglobin levels.