Thomas Bourdrel

TL;DR: Experimental studies indicate that some pollutants have more harmful cardiovascular effects, such as combustion-derived PM2.5 and ultrafine particles, and promotion of safer air quality appears to be a new challenge in cardiovascular disease prevention.

TL;DR: Wang et al. as mentioned in this paper provided evidence from in vitro, animal and human studies from the existing literature, which showed that exposure to air pollution leads to a decreased immune response, thus facilitating viral penetration and replication.

TL;DR: A review of the scientific evidence regarding the effects of the environment on cardiovascular health is presented in this article, where the authors report first influences of air-related environmental factors and conclude that both cold and hot periods have negative cardiovascular impacts, and dynamic changes in temperature are likely more important than the absolute air temperature level.

TL;DR: Cardiovascular impacts of ambient air temperature, indoor and outdoor air pollution, food pollutants, radiation, and some other emerging environmental factors are addressed.

TL;DR: A marked seasonality of NO2, PM2.5, and PM10 concentrations characterized by an increase during winter and a decrease during the summer could be established and the seasonability of coronary events was evidenced simultaneously.