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Thomas Ferraro

Researcher at Rutgers University

Publications -  12
Citations -  2487

Thomas Ferraro is an academic researcher from Rutgers University. The author has contributed to research in topics: Curcumin & Tumor promotion. The author has an hindex of 11, co-authored 12 publications receiving 2449 citations.

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Journal Article

Inhibitory effects of curcumin on in vitro lipoxygenase and cyclooxygenase activities in mouse epidermis.

TL;DR: The inhibitory effects of curcumin, chlorogenic acid, caffeic acid, and ferulic acid on TPA-induced tumor promotion in mouse epidermis parallel their inhibitory effect on T PA-induced epidermal inflammation and epider mal lipoxygenase and cyclo oxygengenase activities.
Journal Article

Inhibition of skin tumorigenesis by rosemary and its constituents carnosol and ursolic acid.

TL;DR: Topical application of carnosol or ursolic acid isolated from rosemary inhibited TPA-induced ear inflammation, ornithine decarboxylase activity, and tumor promotion and applied to mice previously initiated with DMBA inhibited the number of skin tumors per mouse.
Journal Article

Inhibitory effect of green tea in the drinking water on tumorigenesis by ultraviolet light and 12-O-tetradecanoylphorbol-13-acetate in the skin of SKH-1 mice.

TL;DR: Treatment of the animals with the green tea extract not only decreased the number of skin tumors but also decreased substantially the size of the tumors.
Journal ArticleDOI

Inhibitory effect of topical application of a green tea polyphenol fraction on tumor initiation and promotion in mouse skin.

TL;DR: Topical application of green tea polyphenol fraction inhibited TPA-induced inflammation, ornithine decarboxylase activity, hyperplasia and hydrogen peroxide formation, studies with individual polyphenolic compounds in green tea indicated.
Journal Article

Inhibition of Tumor Promoter-mediated Processes in Mouse Skin and Bovine Lens by Caffeic Acid Phenethyl Ester

TL;DR: Findings point to CAPE as being a potent chemopreventive agent, which may be useful in combating diseases with strong inflammatory and/or oxidative stress components, i.e., various types of cancer and possibly cataract development.