T
Thor Willy Ruud Hansen
Researcher at Oslo University Hospital
Publications - 153
Citations - 3206
Thor Willy Ruud Hansen is an academic researcher from Oslo University Hospital. The author has contributed to research in topics: Jaundice & Kernicterus. The author has an hindex of 31, co-authored 150 publications receiving 2870 citations. Previous affiliations of Thor Willy Ruud Hansen include Universiti Sains Malaysia & Rikshospitalet–Radiumhospitalet.
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Journal ArticleDOI
Guidelines for the Management of Extremely Premature Deliveries: A Systematic Review
Úrsula Guillén,Elliott Mark Weiss,David Munson,Pierre Maton,Ann L Jefferies,Mikael Norman,Gunnar Naulaers,Joana Catarina Mendes,Lincoln Justo da Silva,Petr Zoban,Thor Willy Ruud Hansen,Mikko Hallman,Maria Delivoria-Papadopoulos,Shigeharu Hosono,Susan Albersheim,Constance Williams,Elaine M. Boyle,Kei Lui,Brian A Darlow,Haresh Kirpalani +19 more
TL;DR: A systematic review of management from scientific and professional organizations for delivery room care of extremely low GA infants found general agreement for comfort care at 22 weeks’ GA and active care at 25 weeks” GA.
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Neonatal hyperbilirubinaemia: a global perspective.
TL;DR: Up-to-date evidence on the epidemiology of neonatal jaundice including its global burden based on estimates of its prevalence, and both fatal and non-fatal health outcomes is summarized.
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National guidelines for treatment of jaundice in the newborn.
TL;DR: The process employed in Norway to forge agreement on a set of treatment guidelines that are now used across the country are described.
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Bilirubin Has Widespread Inhibitory Effects on Protein Phosphorylation
TL;DR: Results indicate that bilirubin can inhibit catalytic domain(s) of protein kinases by apparent noncompetitive mechanism(s), presumably by interacting with noncatalytic domains on the enzyme.
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Bilirubin and Brain Toxicity
TL;DR: It is suggested that bilirubin encephalopathy should be used in a broader sense so as to include all conditions in which bilirubsin is known, or thought to be, the cause of brain toxicity, to include cases exhibiting the classical acute symptoms described above, or surviving with the typical neurologic sequelae.