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Tomo Suzuki

Researcher at Kyoto Prefectural University of Medicine

Publications -  57
Citations -  2604

Tomo Suzuki is an academic researcher from Kyoto Prefectural University of Medicine. The author has contributed to research in topics: Meibomian gland & Medicine. The author has an hindex of 24, co-authored 44 publications receiving 2287 citations. Previous affiliations of Tomo Suzuki include Harvard University & Massachusetts Eye and Ear Infirmary.

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Cultivated corneal epithelial stem cell transplantation in ocular surface disorders.

TL;DR: C cultivated corneal epithelial transplantation using denuded AM as a carrier can be used for severe stem cell deficiencies and 10 of the 13 eyes were restored to good vision (postoperative visual acuity improved two or more lines) 6 months after the operation.
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Androgen Deficiency, Meibomian Gland Dysfunction, and Evaporative Dry Eye

TL;DR: The results support the hypothesis that androgen deficiency may be an important etiologic factor in the pathogenesis of evaporative dry eye in women with Sjögren's syndrome and demonstrate that the meibomian gland is an androgen target organ.
Journal Article

Cultivated corneal epithelial transplantation for ocular surface reconstruction in acute phase of Stevens-Johnson syndrome.

TL;DR: Corneal stem cell transplantation and amniotic membrane (AM) transplantation, combined with dry eye treatment and strong immunosuppressive therapy, have been attempted at the scarring stage, the management is difficult, and the visual prognosis is not satisfactory.
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Expression of sex steroid hormone receptors in human cornea.

TL;DR: Since receptors for both male and female sex hormones are present in human corneas of both genders, it is postulate that the receptors may influence the biological functions of corneal cells through direct interaction with specific hormones.
Journal Article

Do sex steroids exert sex-specific and/or opposite effects on gene expression in lacrimal and meibomian glands?

TL;DR: The results support the hypothesis that sex steroids may induce sex-specific and/or opposite effects in the lacrimal and meibomian glands and whether these actions contribute to the prevalence of dry eye remains to be determined.