Aquatic chemistry is becoming both a rewarding and substantial area of inquiry and is drawing many prominent scientists to its fold. Its literature has changed from a compilation of compositional tables to studies of the chemical reactions occurring within the aquatic environments. But more than this is the recognition that human society in part is determining the nature of aquatic systems. Since rivers deliver to the world ocean most of its dissolved and particulate components, the interactions of these two sets of waters determine the vitality of our coastal waters. This significant vol ume provides not only an introduction to the dynamics of aquatic chem istries but also identifies those materials that jeopardize the resources of both the marine and fluvial domains. Its very title provides its emphasis but clearly not its breadth in considering natural processes. The book will be of great value to those environmental scientists who are dedicated to keeping the resources of the hydrosphere renewable. As the size of the world population becomes larger in the near future and as the uses of materials and energy show parallel increases, the rivers and oceans must be considered as a resource to accept some of the wastes of society. The ability of these waters and the sediments below them to accommodate wastes must be assessed continually. The key questions relate to the capacities of aqueous systems to carry one or more pollutants."

Metal pollution in the aquatic environment

Advances in metal-induced oxidative stress and human disease

Essentiality Toxicity Carcinogenicity Lead(Pb) Exposure Toxicokinetics Toxicity Neurologic, Neurobehavioral, and Developmental Effects in Children Mechanisms of Effects on the Developing Nervous System Peripheral Neuropathy Hematologic Effects Renal Toxicity Lead and Gout Effects on Cardiovascular System Immunotoxicity Bone Effects Reproductive Effects Birth Outcomes Carcinogenicity Other Effects Dose Response Treatment Organic Lead Compounds Mercury (Hg) Exposure Disposition and Toxicokinetics Metabolic Transformation Cellular Metabolism Toxicology Biological Indicators Treatment Nickel (Ni) Exposure Toxicokinetics Essentiality Toxicity Nickel Carbonyl Poisoning Dermatitis Indicators of Nickel Toxicity

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Toxic effects of metals

https://www.thelancet.com/pdfs/journals/lancet/PIIS0140-6736(06)69665-7.pdf

Developmental neurotoxicity of industrial chemicals

Agricultural waste material as potential adsorbent for sequestering heavy metal ions from aqueous solutions - a review.

A kinetic model of cadmium metabolism in the human being

Cadmium and zinc have been analyzed in tissues from 292 persons autopsied in Stockholm. In kidney cortex, liver, and pancreas the individual cadmium levels are distributed in a lognormal way. In kidney cortex there is a continuous accumulation of cadmium with age up to 50 years, followed by a decrease. Smokers show a higher cadmium accumulation. For nonsmokers, the biological half time of cadmium in kidney cortex is estimated at 30 years, with an average concentration at age 50 of 11 mug/g wet weight. When smokers are included, the average cadmium concentration at age 50 is 22 mug/g wet weight. Based on the more pronounced cadmium accumulation among smokers than nonsmokers, the respiratory absorption rate of cadmium from tobacco smoke is estimated to be approximately 50%.

Cadmium in kidney cortex, liver, and pancreas from Swedish autopsies. Estimation of biological half time in kidney cortex, considering calorie intake and smoking habits.

Studies were carried out in Japan, United States, and Sweden regarding comparability of analytical methods for cadmium, daily intake of cadmium via food, daily amount of cadmium in feces, concentra...

Exposure and accumulation of cadmium in populations from Japan, the United States, and Sweden

Dose-response analysis of cadmium-induced tubular proteinuria: A study of urinary β2-microglobulin excretion among workers in a battery factory

Urinary beta2-microglobulin excretion among people exposed to cadmium in the general environment: an epidemiological study in cooperation between Japan and Sweden.

Tord Kjellström

Papers

A kinetic model of cadmium metabolism in the human being

Cadmium in kidney cortex, liver, and pancreas from Swedish autopsies. Estimation of biological half time in kidney cortex, considering calorie intake and smoking habits.

Exposure and accumulation of cadmium in populations from Japan, the United States, and Sweden

Dose-response analysis of cadmium-induced tubular proteinuria: A study of urinary β2-microglobulin excretion among workers in a battery factory

Urinary beta2-microglobulin excretion among people exposed to cadmium in the general environment: an epidemiological study in cooperation between Japan and Sweden.