T
Travis J. Hollmann
Researcher at Brigham and Women's Hospital
Publications - 6
Citations - 1015
Travis J. Hollmann is an academic researcher from Brigham and Women's Hospital. The author has contributed to research in topics: Histone methyltransferase & Chromatin immunoprecipitation. The author has an hindex of 5, co-authored 6 publications receiving 872 citations.
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Journal ArticleDOI
The histone methyltransferase SETDB1 is recurrently amplified in melanoma and accelerates its onset
Craig J. Ceol,Yariv Houvras,Yariv Houvras,Yariv Houvras,Judit Jané-Valbuena,Judit Jané-Valbuena,Steve Bilodeau,David A. Orlando,Valentine Battisti,Lauriane Fritsch,William M. Lin,William M. Lin,Travis J. Hollmann,Fabrizio Ferrè,Caitlin Bourque,Christopher J. Burke,Laura Turner,Audrey Uong,Laura A. Johnson,Laura A. Johnson,Rameen Beroukhim,Rameen Beroukhim,Craig H. Mermel,Craig H. Mermel,Massimo Loda,Slimane Ait-Si-Ali,Levi A. Garraway,Levi A. Garraway,Richard A. Young,Leonard I. Zon +29 more
TL;DR: SETDB1, an enzyme that methylates histone H3 on lysine 9 (H3K9), was found to accelerate melanoma formation significantly in zebrafish and is established as an oncogene in melanoma and underscores the role of chromatin factors in regulating tumorigenesis.
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INI1-deficient tumors: diagnostic features and molecular genetics
TL;DR: The clinicopathologic features of these tumor types are reviewed and the clinical utility of INI1 immunohistochemistry in differential diagnosis is emphasized, especially in relation to rhabdoid cytomorphology.
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Digital fibromyxoma (superficial acral fibromyxoma): a detailed characterization of 124 cases.
TL;DR: Sequencing of 8 digital fibromyxomas failed to reveal mutations in exon 8 or 9 of GNAS1, in contrast to intramuscular or cellular myxoma, and all tumors lacked necrosis, pleomorphism, or neural/perineural infiltration.
Journal ArticleDOI
CD25 expression on cutaneous mast cells from adult patients presenting with urticaria pigmentosa is predictive of systemic mastocytosis.
TL;DR: It is suggested that immunoreactivity for CD25 in cutaneous mast cells may be useful for stratifying adult patients presenting with UP for additional clinical evaluation.
The SETDB1 histone methyltransferase is recurrently amplified in and accelerates melanoma
Craig Ceol,Yariv Houvras,Judit Jané-Valbuena,Steve Bilodeau,David A. Orlando,Valentine Battisti,Lauriane Fritsch,William M. Lin,Travis J. Hollmann,Fabrizio Ferrè,Caitlin Bourque,Christopher J. Burke,Laura Turner,Audrey Uong,Laura A. Johnson,Rameen Beroukhim,Craig H. Mermel,Massimo Loda,Slimane Ait-Si-Ali,Levi A. Garraway,Richard A. Young,Leonard I. Zon +21 more
TL;DR: SETDB1, an enzyme that methylates histone H3 on lysine 9 (H3K9), was found to significantly accelerate melanoma formation in the zebrafish and established SETDB1 as an oncogene in melanoma and underscore the role of chromatin factors in regulating tumorigenesis.