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Valeria Rettori

Researcher at University of Buenos Aires

Publications -  63
Citations -  2671

Valeria Rettori is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Hypothalamus & Luteinizing hormone. The author has an hindex of 30, co-authored 63 publications receiving 2604 citations. Previous affiliations of Valeria Rettori include University of Texas Southwestern Medical Center.

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Role of nitric oxide in the control of luteinizing hormone-releasing hormone release in vivo and in vitro

TL;DR: The combined in vivo and in vitro results indicate that the pulsatile release of LHRH induced by norepinephrine is brought about by alpha 1-adrenergic activation of NO synthase, which induces prostaglandin E2 release that activates exocytosis of L HRH secretory granules into the portal vessels to induce pulsatile LH release.
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Nitric oxide mediates norepinephrine-induced prostaglandin E2 release from the hypothalamus.

TL;DR: The indication that NO is essential to norepinephrine-induced release of PGE2 from hypothalamic fragments provides insight into the mechanism of LHRH release and the results open the possibility that the importance of NO to neuronal functions may be widespread in the nervous system.
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Role of nitric oxide in control of prolactin release by the adenohypophysis.

TL;DR: No appears to play little role in the prolactin-releasing action of vasoactive intestinal polypeptide and substance P, but mediates the prolACTin-inhibiting activity of dopamine and atrial natriuretic factor.
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Cachectin alters anterior pituitary hormone release by a direct action in vitro

TL;DR: The results suggest that at least part of the stimulatory action of the peptide on pituitary hormone release is brought about by prostaglandins.
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Physiologically Significant Effect of Neuropeptide Y to Suppress Growth Hormone Release by Stimulating Somatostatin Discharge

TL;DR: NPY has a physiologically significant inhibitory action within the hypothalamus to suppress GH release in ovariectomized female and intact male rats by stimulation of SRIF release by alpha 1 and beta-adrenergic receptor-mediated mechanisms.