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Vera L. Petricevich

Researcher at Universidad Autónoma del Estado de Morelos

Publications -  49
Citations -  1470

Vera L. Petricevich is an academic researcher from Universidad Autónoma del Estado de Morelos. The author has contributed to research in topics: Tumor necrosis factor alpha & Venom. The author has an hindex of 22, co-authored 45 publications receiving 1281 citations. Previous affiliations of Vera L. Petricevich include Instituto Butantan.

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Scorpion Venom and the Inflammatory Response

TL;DR: In models of severe systemic inflammation produced by injection of high doses of venom or venoms products, the increase in production of proinflammatory cytokines significantly contributes to immunological imbalance, multiple organ dysfunction and death.
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Increments in serum cytokine and nitric oxide levels in mice injected with Bothrops asper and Bothrops jararaca snake venoms

TL;DR: A complex pattern of cytokine and nitric oxide synthesis and secretion occurs in severe experimental envenomation by B. asper and B. jararaca venoms, and it is suggested that some of these mediators might play a relevant role in the pathophysiology of systemic alterations induced by these venoms.
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Characterization of Chemical Compounds with Antioxidant and Cytotoxic Activities in Bougainvillea x buttiana Holttum and Standl, (var. Rose) Extracts.

TL;DR: The results show that the highest extraction yield was observed with water and meethanol, and the maximum total phenolic content amount and highest antioxidant potential were obtained when extraction with methanol was used.
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Local inflammation, lethality and cytokine release in mice injected with Bothrops atrox venom

TL;DR: The results suggest that susceptibility to Bothrops atrox venom is genetically dependent but MHC independent; that IL-6, IL-10, TNF-alpha, IFN-gamma and NO can be involved in the mediation of tissue damage; and that the major venom component inducers of the lesions are haemorrhagins.
Journal Article

Incorporation of a 35-kilodalton purified protein from Loxosceles intermedia spider venom transforms human erythrocytes into activators of autologous complement alternative pathway.

TL;DR: The data suggest that the C system mediates the lysis of human erythrocytes and, by extension, of other cell types able to incorporate the lytic factor of Loxosceles venoms on their cell surfaces.