V
Verne Nelson
Researcher at March of Dimes
Publications - 20
Citations - 3282
Verne Nelson is an academic researcher from March of Dimes. The author has contributed to research in topics: Population & Pregnancy. The author has an hindex of 16, co-authored 20 publications receiving 2857 citations. Previous affiliations of Verne Nelson include California Health and Human Services Agency.
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Journal ArticleDOI
Maternal pesticide exposure from multiple sources and selected congenital anomalies.
TL;DR: Investigating multiple sources of potential pesticide exposures without more specific information on chemical and level of exposure could not adequately discriminate whether the observed effects are valid, whether biased exposure reporting contributed to the observed elevated risks, or whether nonspecific measurement of exposure was responsible for many of the observed estimated risks not being elevated.
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Hypospadias in California: trends and descriptive epidemiology.
Suzan L. Carmichael,Gary M. Shaw,Verne Nelson,Steve Selvin,Claudine P. Torfs,Cynthia J. Curry +5 more
TL;DR: This study suggests that hypospadias prevalence has not been increasing in California in recent years and differences by phenotype suggest that examining certain phenotypes separately could help to understand hypos padias etiology.
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Schizencephaly: heterogeneous etiologies in a population of 4 million California births.
TL;DR: It is suggested that schizencephaly has heterogeneous etiologies many of which are vascular disruptive in origin, including gastroschisis, bowel atresias, and amniotic band disruption sequence.
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Neural tube defects associated with maternal periconceptional dietary intake of simple sugars and glycemic index
Gary M. Shaw,Thu Quach,Verne Nelson,Suzan L. Carmichael,Donna M. Schaffer,Steve Selvin,Wei Yang +6 more
TL;DR: The observed associations support observations that potential problems in glucose control are associated with NTD risk even among nondiabetic women.
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Socio-economic status and risk of conotruncal heart defects and orofacial clefts.
TL;DR: It is suggested that SES risks are birth defect specific, and low SES was associated with increased risk of d-transposition of the great arteries (dTGA), reduced risk of tetralogy of Fallot (TOF), but was not associated with risk of orofacial clefts.