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Vicente Molina

Researcher at University of Valladolid

Publications -  111
Citations -  2405

Vicente Molina is an academic researcher from University of Valladolid. The author has contributed to research in topics: Schizophrenia & Psychosis. The author has an hindex of 27, co-authored 103 publications receiving 2171 citations. Previous affiliations of Vicente Molina include University of Salamanca & Spanish National Research Council.

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Influence of the normalization template on the outcome of statistical parametric mapping of PET scans

TL;DR: Results show that the use of the standard template under inappropriate conditions (different tracer or mental state) may lead to inconsistent interpretations of the statistical analysis.
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Increase in gray matter and decrease in white matter volumes in the cortex during treatment with atypical neuroleptics in schizophrenia.

TL;DR: Treatment with risperidone and clozapine in schizophrenia may have an effect on gray and white matter volume and needs further exploration.
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Anatomical and functional brain variables associated with clozapine response in treatment-resistant schizophrenia.

TL;DR: Clinical improvement with clozapine may be related with the anatomy and metabolic activity of specific brain areas, with the structural integrity of the DLPF and temporal regions showing the maximum predictive capacity.
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Cerebral metabolic changes induced by clozapine in schizophrenia and related to clinical improvement

TL;DR: Results show that haloperidol and clozapine produce different patterns of metabolic changes in schizophrenia, and the improvement in disorganization, negative and positive syndromes with clozabine may be respectively associated with metabolicChanges in the motor area, basal ganglia, and visual cortex.
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Auditory P300 event related potential and serotonin reuptake inhibitor treatment in obsessive-compulsive disorder patients.

TL;DR: It is suggested that cognitive dysfunction in OCD fluctuates with changes in the clinical associated with treatment, probably in relationship to central serotoninergic transmission.