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Viol Dhea Kharisma

Researcher at University of Brawijaya

Publications -  51
Citations -  482

Viol Dhea Kharisma is an academic researcher from University of Brawijaya. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 4, co-authored 23 publications receiving 66 citations.

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Journal ArticleDOI

Herbal combination from Moringa oleifera Lam. and Curcuma longa L. as SARS-CoV-2 antiviral via dual inhibitor pathway: A viroinformatics approach

TL;DR: In this paper , a combination of Moringa oleifera Lam and Curcuma longa L. is predicted to have antiviral and anti-inflammatory activity through dual inhibitor mechanism played by cryptochlorogenic acid and curcumin.
Book ChapterDOI

Retrieval and Investigation of Data on SARS-CoV-2 and COVID-19 Using Bioinformatics Approach.

TL;DR: In this article, the authors report essential bioinformatics entities, such as database and platform development, molecular evolution and phylogenetic analyses, and vaccine designs, that are useful to solve the SARS-CoV-2 conundrum.
Journal ArticleDOI

Genetic variant of SARS-CoV-2 isolates in Indonesia: Spike glycoprotein gene

TL;DR: Interestingly, there were no significant changes in the genetic composition of SARS-CoV-2 spike glycoprotein genes, when compared to the Wuhan- Hu-1 isolate from China, however, this is a preliminary study and it is recommended that molecular epidemiology and surveillance programs against COVID-19 in Indonesia be improved.
Journal ArticleDOI

Distribution of COVID-19 and phylogenetic tree construction of sars-CoV-2 in Indonesia

TL;DR: The distribution of COVID-19 in Indonesia and the SARS-CoV-2 phylogenetic tree from Indonesian isolates and those from other countries, including other CoVs to determine their relationship were described.
Journal ArticleDOI

Construction of epitope-based peptide vaccine against SARS-CoV-2: Immunoinformatics study

TL;DR: Interestingly, Pep-4 ADHQPQTFVNTELH is recommended as a epitope-based peptide vaccine candidate to deal with the SARS-CoV-2 outbreak, which has a high level of immunogenicity and does not trigger autoimmune mechanisms.