W.J. Slierendrecht

TL;DR: It is demonstrated that additive genetic (co)variation for growth rate, feed conversion efficiency, and VHS resistance does exist within the farmed population of rainbow trout, and indicates that selective breeding for these traits can be successful.

TL;DR: The additive genetic variation detected for resistance demonstrates that selectively breeding trout for resistance to ERM, RTFS, and VHS will be successful, providing a complementary approach to control these diseases.

TL;DR: The susceptibility of rainbow trout Oncorhynchus mykiss to infection with various isolates of viral haemorrhagic septicaemia virus (VHSV) was examined, supporting the view that they originated from the marine environment.

TL;DR: A monoclonal antibody against rainbow trout peripheral blood leucocytes was selected for its lack of reactivity with rainbow trout immunoglobulin and revealed that the antibody reacted strongly with granulocytes and weakly with thrombocytes, and not with erythrocytes, lymphocytes, monocytes or macrophages.

TL;DR: An attempt to associate the distribution of different MHC class II genotypes with low and high resistance gynogenetic offspring was performed, and a significantly different distribution of haplotypes in the low resistance offspring was observed, that could not be explained by a one- or two-locus model.