W
Warren G. Guntheroth
Researcher at University of Washington
Publications - 183
Citations - 4616
Warren G. Guntheroth is an academic researcher from University of Washington. The author has contributed to research in topics: Sudden infant death syndrome & Pulmonary artery. The author has an hindex of 35, co-authored 183 publications receiving 4523 citations. Previous affiliations of Warren G. Guntheroth include University of Massachusetts Medical School.
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The hemodynamics of cardiac tamponade and constrictive pericarditis.
TL;DR: Both constrictive pericarditis and cardiac tamponade increase pulmonary and systemic venous pressure and decrease cardiac output and stroke volume and pulsus paradoxus is almost invariably present.
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How important are dental procedures as a cause of infective endocarditis
TL;DR: Although oral chemoprophylaxis for major dental procedures appears prudent, the British regimen of a single dose of amoxicillin administered orally is much simpler and probably more effective, and scrupulous oral and dental hygiene is undoubtedly superior in preventing IE.
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Hemodynamic Effects of Intermittent Positive Pressure Respiration
Beverly C. Morgan,Wayne E. Martin,Thomas F. Hornbein,Edward W. Crawford,Warren G. Guntheroth +4 more
TL;DR: The hemodynamic effects of intermittent positive pressure ventilation were studied in lightly anesthetized dogs following recovery from implantation of pulsed ultrasonic flow transducers on the aorta and vena cava to study the circulatory effects of positive pressure breathing and the effect on venous return.
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The triple risk hypotheses in sudden infant death syndrome.
TL;DR: Sudden infant death syndrome (SIDS) victims were regarded as normal as a matter of definition until 1952 when Kinney and colleagues argued for elimination of the clause, "unexpected by history."
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Flow patterns in cavae, pulmonary artery, pulmonary vein, and aorta in intact dogs
TL;DR: Flow patterns in cavae, pulmonary artery, pulmonary vein, and aorta in intact dogs were remarkably similar to caval flow, and in the absence of transmitted pulmonary artery pressure pulses, the venous flow patterns are attributed to vis a fronte.