W
Wei Yang
Researcher at Stanford University
Publications - 107
Citations - 3008
Wei Yang is an academic researcher from Stanford University. The author has contributed to research in topics: Population & Pregnancy. The author has an hindex of 29, co-authored 96 publications receiving 2493 citations. Previous affiliations of Wei Yang include California Health and Human Services Agency & March of Dimes.
Papers
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Journal ArticleDOI
118 SNPs of folate-related genes and risks of spina bifida and conotruncal heart defects
Gary M. Shaw,Gary M. Shaw,Gary M. Shaw,Wei Lu,Huiping Zhu,Wei Yang,Farren B.S. Briggs,Suzan L. Carmichael,Suzan L. Carmichael,Lisa F. Barcellos,Edward J. Lammer,Richard H. Finnell +11 more
TL;DR: The authors' observations do not implicate a particular folate transport or metabolism gene to be strongly associated with risks for spina bifida or conotruncal defects and multiple SNPs observed for a given gene showed evidence of linkage disequilibrium indicating that the observed SNPs were not individually contributing to risk.
Journal ArticleDOI
Maternal Food Insecurity Is Associated with Increased Risk of Certain Birth Defects
TL;DR: The study suggests that increased risks of certain birth defects may be included among the negative consequences of food insecurity, and several models suggested effect modification by certain factors.
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Orofacial clefts in the National Birth Defects Prevention Study, 1997–2004
Alicia E. Genisca,Jaime L. Frias,Cheryl S. Broussard,Margaret A. Honein,Edward J. Lammer,Cynthia A. Moore,Gary M. Shaw,Jeffrey C. Murray,Wei Yang,Sonja A. Rasmussen +9 more
TL;DR: Among infants with CL or CLP, heart, limb, and other musculoskeletal defects were most commonly observed, while heart, limbs, and central nervous system defect were most common among infants with CP.
Journal ArticleDOI
Epidemiologic characteristics of congenital diaphragmatic hernia among 2.5 million California births, 1989-1997.
TL;DR: The authors' observations add to the relatively few population-based descriptive epidemiologic studies of the prevalence and mortality, and maternal and infant characteristics of CDH.
Journal ArticleDOI
Ambient air pollution and traffic exposures and congenital heart defects in the San Joaquin Valley of California.
Amy Padula,Ira B. Tager,Suzan L. Carmichael,Suzan L. Carmichael,S. Katharine Hammond,Wei Yang,Frederick Lurmann,Gary M. Shaw +7 more
TL;DR: PM10 and traffic density may contribute to the occurrence of pulmonary valve stenosis and ventricular septal defects, respectively.