W
Weiwei Liu
Researcher at Zunyi Medical College
Publications - 13
Citations - 293
Weiwei Liu is an academic researcher from Zunyi Medical College. The author has contributed to research in topics: Medicine & Microvesicles. The author has an hindex of 4, co-authored 7 publications receiving 117 citations.
Papers
More filters
Journal ArticleDOI
Exosomal circHIPK3 Released from Hypoxia-Pretreated Cardiomyocytes Regulates Oxidative Damage in Cardiac Microvascular Endothelial Cells via the miR-29a/IGF-1 Pathway.
Yan Wang,Ranzun Zhao,Weiwei Liu,Zhenglong Wang,Jidong Rong,Xianping Long,Zhijiang Liu,Junbo Ge,Bei Shi +8 more
TL;DR: CircHIPK3 in HPC-exos plays a role in CMVECs under oxidative conditions through miR-29a-mediated IGF-1 expression, leading to a decrease in oxidative stress-induced CMV ECs dysfunction.
Journal ArticleDOI
Exosomes Derived from miR-214-Enriched Bone Marrow-Derived Mesenchymal Stem Cells Regulate Oxidative Damage in Cardiac Stem Cells by Targeting CaMKII.
Yan Wang,Ranzun Zhao,Debin Liu,Wenwen Deng,Guanxue Xu,Weiwei Liu,Jidong Rong,Xianping Long,J.B Ge,Bei Shi +9 more
TL;DR: It is suggested that BMSCs release miR-214-containing exosomes to suppress oxidative stress injury in CSCs through CaMKII silencing, which regulates oxidative stress-related injury in BMSC-exos.
Journal ArticleDOI
CircUbe3a from M2 macrophage-derived small extracellular vesicles mediates myocardial fibrosis after acute myocardial infarction.
Yan Wang,Chaofu Li,Ranzun Zhao,Zhimei Qiu,Changyin Shen,Zhenglong Wang,Weiwei Liu,Wei Zhang,Junbo Ge,Bei Shi +9 more
TL;DR: In this paper, the role of circular RNAs (circRNAs) in M2 macrophage (M2M)-derived small extracellular vesicles (SEVs) in myocardial fibrosis development was explored.
Journal ArticleDOI
Exosomal CircHIPK3 Released from Hypoxia-Induced Cardiomyocytes Regulates Cardiac Angiogenesis after Myocardial Infarction.
Yan Wang,Ranzun Zhao,Changyin Shen,Weiwei Liu,Jinson Yuan,Chaofu Li,Wenwen Deng,Zhenglong Wang,Wei Zhang,Junbo Ge,Bei Shi +10 more
TL;DR: This study provides a novel mechanism by which exosomal circRNAs are involved in the communication between CMs and cardiac endothelial cells and reports that hypoxic exosomes (HPC-exos) can effectively reduce theinfarct area and promote angiogenesis in the border surrounding the infarcted area.
Journal ArticleDOI
CircHIPK3 regulates the autophagy and apoptosis of hypoxia/reoxygenation-stimulated cardiomyocytes via the miR-20b-5p/ATG7 axis.
Zhimei Qiu,Yan Wang,Weiwei Liu,Chaofu Li,Ranzun Zhao,Xianping Long,Jidong Rong,Wengweng Deng,Changyin Shen,Jinson Yuan,Wengming Chen,Bei Shi +11 more
TL;DR: In this article, the authors examined the regulatory effect of circHIPK3 during myocardial ischemia/reperfusion (I/R) injury and investigated its mechanism in cardiomyocyte autophagy and apoptosis.