William M. U. Daniels

TL;DR: Depressive-like behavior, measured during a forced swim test, was seen in maternally separated rats after additional chronic stress during adulthood, possibly as an effect of high corticosterone levels, but compensatory mechanisms against cell death may be involved as neurotrophin levels increased in the dorsal hippocampus.

TL;DR: Compared with melatonin in an in vitro system which measures lipid peroxidation of platelet membranes in the presence of iron, cholesterol and beta-sitosterol increased lipidPeroxidation, while DHEAS and melatonin continued to decrease lipid per oxidation.

TL;DR: Prenatal stress appeared to enhance the toxic effect of 6-OHDA possibly by reducing the compensatory adaptations to exercise and voluntary exercise appeared to have a neuroprotective effect resulting in an improvement in motor control and decreased asymmetry in the use of left and right forelimbs to explore a novel environment.

TL;DR: Serotonin exerts its free radical scavenging action in the aqueous phase, or at the water-membrane interface, while melatonin positions itself within the lipid bilayer where it protects membrane phospholipids against free radical attack.

TL;DR: The results indicate that chronic administration of CRF into the basolateral amygdala may promote stress-induced grooming behavior in rats and suggests that increased CRF in the amygdala may contribute to the dysregulation of corticosterone secretion.