Winfried Mohr

TL;DR: In this article, the tissue architecture of synovial membranes obtained from patients with non-inflammatory joint diseases and patients with rheumatoid arthritis was studied using monoclonal reagents directed towards antigens of the monocyte-macrophage lineage and Ia antigen.

TL;DR: The tissue architecture of synovial membranes obtained from patients with non-inflammatory joint diseases and patients with rheumatoid arthritis was studied, focusing on the localization of the type I, type II and type III synoviocytes that previously had been defined by their cell surface phenotype.

TL;DR: Commercial pure Ti should be treated as the preferred material for osteosyntheses if a removal of the implant is not intended, however, neither material provoked a foreign-body reaction in the local tissues, thus cpTi cannot be recommend as the ‘golden standard’ for osteOSynthesis material in general.

TL;DR: PMN were encountered more or less often in the pannus but clearly accumulated in a significant amount at the pANNus-cartilage junction in one case of rheumatic monarthritis and three out of four cases with rheumatoid arthritis, and directly supports the hypothesis that PMN enzymes play an active role in rheumatism.

TL;DR: It is concluded that PMNs in the inflammatory synovial fluid may gain access to the fibrous structures of the joint, thus participating in tissue destruction, as has been assumed from in vitro investigations by other authors.