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Xing-Sheng Ren

Researcher at Nanjing Medical University

Publications -  16
Citations -  814

Xing-Sheng Ren is an academic researcher from Nanjing Medical University. The author has contributed to research in topics: Vascular smooth muscle & Angiotensin II. The author has an hindex of 13, co-authored 14 publications receiving 560 citations.

Papers
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Journal ArticleDOI

NLRP3 inflammasome activation contributes to VSMC phenotypic transformation and proliferation in hypertension.

Hai-Jian Sun, +11 more
- 05 Oct 2017 - 
TL;DR: Results indicate that NLRP3 inflammasome activation response to histone acetylation and NFκB activation contributes to VSMC phenotype switching and proliferation and vascular remodeling in hypertension.
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FNDC5 Alleviates Hepatosteatosis by Restoring AMPK/mTOR-Mediated Autophagy, Fatty Acid Oxidation and Lipogenesis in Mice.

Tong-Yan Liu, +13 more
- 08 Aug 2016 - 
TL;DR: It is indicated that FNDC5 deficiency impairs autophagy and FAO and enhances lipogenesis via the AMPK/mTOR pathway and prevents the HFD-induced hyperlipemia, hepatic lipid accumulation, and impairedFAO and Autophagy in the liver.
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Angiotensin-(1–7) abrogates angiotensin II-induced proliferation, migration and inflammation in VSMCs through inactivation of ROS-mediated PI3K/Akt and MAPK/ERK signaling pathways

Feng Zhang, +4 more
- 30 Sep 2016 - 
TL;DR: Results indicate that Ang-(1–7) antagonizes the Ang II-induced VSMC proliferation, migration and inflammation through activation of Mas receptor and then suppression of ROS-dependent PI3K/Akt and MAPK/ERK signaling pathways.
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Salusin-β Promotes Vascular Smooth Muscle Cell Migration and Intimal Hyperplasia After Vascular Injury via ROS/NFκB/MMP-9 Pathway.

Hai-Jian Sun, +8 more
- 21 Jun 2016 - 
TL;DR: It is proposed that salusin-β may be important in the VSMC migration and neointima of some vascular diseases.
Journal ArticleDOI

NLRP3 Gene Deletion Attenuates Angiotensin II-Induced Phenotypic Transformation of Vascular Smooth Muscle Cells and Vascular Remodeling.

Xing-Sheng Ren, +10 more
- 01 Jan 2017 - 
TL;DR: NLRP3 inflammasome activation contributes to Ang II-induced VSMC phenotypic transformation and proliferation as well as vascular remodeling and hypertension.