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Yasuyuki Yamaji

Researcher at University of Tokyo

Publications -  115
Citations -  3056

Yasuyuki Yamaji is an academic researcher from University of Tokyo. The author has contributed to research in topics: Gene & Plant virus. The author has an hindex of 29, co-authored 103 publications receiving 2531 citations.

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A unique virulence factor for proliferation and dwarfism in plants identified from a phytopathogenic bacterium

TL;DR: It is shown that a single virulence factor, tengu-su inducer (TENGU), induces witches' broom and dwarfism and is a small secreted protein of the plant-pathogenic bacterium, phytoplasma.
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Interaction between the membrane protein of a pathogen and insect microfilament complex determines insect-vector specificity

TL;DR: This study focused on an abundant cell-surface membrane protein of the phytoplasma named antigenic membrane protein (Amp), which is not homologous with any reported functional protein, and reported interactions between pathogens and mammalian microfilaments between a bacterial surface protein and a host microfilament in insect cells.
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Recessive Resistance to Plant Viruses: Potential Resistance Genes Beyond Translation Initiation Factors.

TL;DR: This review focuses on recent advances related to antiviral recessive resistance genes evaluated in model plants and several crop species, and addresses the roles of next-generation sequencing and genome editing technologies in improving plant genetic resources for recessive Resistance-based antiviral breeding in various crop species.
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Lectin-Mediated Resistance Impairs Plant Virus Infection at the Cellular Level

TL;DR: Analysis of resistance of certain Arabidopsis thaliana ecotypes to a potexvirus to show the existence of lectin genes that show a variety of levels of virus resistance, their targets, and their properties, which are distinct from those of known R genes, suggests the generality of Lectin-mediated resistance in plant innate immunity.
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Viral-induced systemic necrosis in plants involves both programmed cell death and the inhibition of viral multiplication, which are regulated by independent pathways.

TL;DR: It is suggested that systemic necrosis and HR consist of PCD and a restraint of virus multiplication, and that the latter is induced through unknown pathways independent from the former.