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Yongli Xuan
Researcher at Shandong University
Publications - 15
Citations - 253
Yongli Xuan is an academic researcher from Shandong University. The author has contributed to research in topics: Cardiac nerve & Cardiac function curve. The author has an hindex of 10, co-authored 13 publications receiving 204 citations.
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Journal ArticleDOI
Inhibition of Notch signaling pathway attenuates sympathetic hyperinnervation together with the augmentation of M2 macrophages in rats post-myocardial infarction
Jie Yin,Hesheng Hu,Xiaolu Li,Mei Xue,Wenjuan Cheng,Ye Wang,Yongli Xuan,Xinran Li,Na Yang,Yugen Shi,Suhua Yan +10 more
TL;DR: It is concluded that Notch inhibition during the acute inflammatory response phase is associated with the downregulation of NGF, probably through a macrophage-dependent pathway, thus preventing the process of sympathetic hyperinnervation.
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Targeted NGF siRNA Delivery Attenuates Sympathetic Nerve Sprouting and Deteriorates Cardiac Dysfunction in Rats with Myocardial Infarction
Hesheng Hu,Yongli Xuan,Ye Wang,Mei Xue,Fei Suo,Xiaolu Li,Wenjuan Cheng,Xinran Li,Jie Yin,Ju Liu,Suhua Yan +10 more
TL;DR: It is suggested that endogenous NGF silencing attenuated sympathetic nerve sprouting and angiogenesis, enlarged the infarct size, aggravated cardiac dysfunction, and potentially contributed to an unfavorable prognosis after myocardial infarction.
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Risk of ventricular arrhythmias after myocardial infarction with diabetes associated with sympathetic neural remodeling in rabbits.
TL;DR: MI results in sympathetic neural remodeling in diabetic rabbits, which may be responsible in part for the increased occurrence of VAs.
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Semaphorin 3A attenuates cardiac autonomic disorders and reduces inducible ventricular arrhythmias in rats with experimental myocardial infarction.
Hesheng Hu,Yongli Xuan,Mei Xue,Wenjuan Cheng,Ye Wang,Xinran Li,Jie Yin,Xiaolu Li,Na Yang,Yugen Shi,Suhua Yan +10 more
TL;DR: The upregulation and administration of sema 3A exerted beneficial effects on infarction-induced cardiac autonomic disorders by increasing cardiac electrical stability and reducing VAs.
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Atorvastatin attenuates sympathetic hyperinnervation together with the augmentation of M2 macrophages in rats postmyocardial infarction.
Na Yang,Wenjuan Cheng,Hesheng Hu,Mei Xue,Xiaolu Li,Ye Wang,Yongli Xuan,Xinran Li,Jie Yin,Yugen Shi,Suhua Yan +10 more
TL;DR: Atorvastatin effectively ameliorated cardiac sympathetic nerve remodeling and prevented VAs after MI by significantly downregulating the expression of NGF, IL-1β, and TNF-α via together with the augmentation of M2 macrophage.