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Youn-Chul Kim

Researcher at Wonkwang University

Publications -  339
Citations -  9394

Youn-Chul Kim is an academic researcher from Wonkwang University. The author has contributed to research in topics: Heme oxygenase & Nitric oxide synthase. The author has an hindex of 48, co-authored 339 publications receiving 8272 citations. Previous affiliations of Youn-Chul Kim include Woosuk University.

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Sauchinone suppresses pro-inflammatory mediators by inducing heme oxygenase-1 in RAW264.7 macrophages.

TL;DR: Results suggest that sauchinone inhibits pro-inflammatory mediators through expression of anti-inflammatory HO-1 via ERK pathway.
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Hericirine, a novel anti-inflammatory alkaloid from Hericium erinaceum

TL;DR: In this article, a novel ergosterol conjunction-type alkaloid, hericirine (1 ), was isolated from the dried fruiting bodies of Hericium erinaceum.
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Cyclic Adenosine Monophosphate Inhibits Ursolic Acid-Induced Apoptosis via Activation of Protein Kinase A in Human Leukaemic HL-60 Cells: cAMP ON URSOLIC ACID-INDUCED APOPTOSIS

TL;DR: The results suggest that signaling pathway of cAMP-dependent activation of protein kinase A may affect the responsiveness of tumor cells upon ursolic acid.
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Simultaneous determination of tanshinone I, dihydrotanshinone I, tanshinone IIA and cryptotanshinone in rat plasma by liquid chromatography-tandem mass spectrometry: application to a pharmacokinetic study of a standardized fraction of Salvia miltiorrhiza, PF2401-SF.

TL;DR: A rapid, sensitive and selective liquid chromatography-tandem mass spectrometric (LC-MS/MS) method for the simultaneous determination of tanshinone I, dihydrotanshin one I, tansinone IIA and cryptotanshinOne, the active components of Salvia miltiorrhiza in rat plasma, was developed.
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Inhibitory effects of butanol fraction of the aqueous extract of Forsythia koreana on the nitric oxide production by murine macrophage-like RAW 264.7 cells

TL;DR: Findings suggest that inhibition of NO production by this butanol fraction in RAW 264.7 cells stimulated with IFN-gamma plus LPS was due to the suppression of iNOS gene expression.